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Avigail T. Erlich,Liam D. Tryon,Matthew J. Crilly,Jonathan M. Memme,Zahra S. Mesbah Moosavi,Ashley N. Oliveira,Kaitlyn Beyfuss,David A. Hood 한국한의학연구원 2016 Integrative Medicine Research Vol.5 No.3
Skeletal muscle mitochondrial content and function are regulated by a number of specialized molecular pathways that remain to be fully defined. Although a number of proteins have been identified to be important for the maintenance of mitochondria in quiescent muscle, the requirement for these appears to decrease with the activation of multiple overlapping signaling events that are triggered by exercise. This makes exercise a valuable therapeutic tool for the treatment of mitochondrially based metabolic disorders. In this review, we summarize some of the traditional and more recently appreciated pathways that are involved in mitochondrial biogenesis in muscle, particularly during exercise.
Recent advances in mitochondrial turnover during chronic muscle disuse
Liam D. Tryon,Anna Vainshtein,Jonathan M. Memme,Matthew J. Crilly,David A. Hood 한국한의학연구원 2014 Integrative Medicine Research Vol.3 No.4
Chronic muscle disuse, such as that resulting from immobilization, denervation, or prolonged physical inactivity, produces atrophy and a loss of mitochondria, yet the molecular relationship between these events is not fully understood. In this review we attempt to identify the key regulatory steps mediating the loss of muscle mass and the decline in mitochondrial content and function. An understanding of common intracellular signaling pathways may provide much-needed insight into the possible therapeutic targets for treatments that will maintain aerobic energy metabolism and preserve muscle mass during disuse conditions.