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연약해성점토의 깊이별 흡수력 시험과 일축압축시험 결과를 이용한 시료교란도 연구
鄭庸宇,柳完圭,金昞逸,李萬洙 명지대학교 산업기술연구소 2006 産業技術硏究所論文集 Vol.25 No.-
It is important to know the degree of disturbance when evaluating the soil strength parameters from laboratory test. In this study, suction tests and unconfined compression strength tests were carried out to investigate the degree of disturbance of sample with soft marine clay of Kwangyang. Based on the test results, suction test is a good quantitative analysis for the degree of disturbance and shows a similar tendency with the results of unconfined compression strength test.
Lee, Mihye,Paik, Sang Kyoo,Lee, Min-Jung,Kim, Yoon-Jung,Kim, Sungdae,Nahm, Minyeop,Oh, Soo-Jin,Kim, Hyun-Man,Yim, Jeongbin,Lee, C. Justin,Bae, Yong Chul,Lee, Seungbok Elsevier 2009 Developmental Biology Vol.330 No.2
<P><B>Abstract</B></P><P>Hereditary spastic paraplegia (HSP) is an inherited neurological disorder characterized by progressive spasticity and weakness of the lower extremities. The most common early-onset form of HSP is caused by mutations in the human gene that encodes the dynamin-family GTPase Atlastin-1 (Atl-1). Recently, loss of the <I>Drosophila</I> ortholog of Atl-1 (Atl) has been found to induce locomotor impairments from the earliest adult stages, suggesting the developmental role of atlastin-subfamily GTPases. Here, we provide evidence that Atl is required for normal growth of muscles and synapses at the neuromuscular junction (NMJ). Atl protein is highly expressed in larval body-wall muscles. Loss-of-function mutations in the <I>atl</I> gene reduce the size of muscles and increase the number of synaptic boutons. Rescue of these defects is accomplished by muscular, but not neuronal expression of Atl. Loss of Atl also disrupts ER and Golgi morphogenesis in muscles and reduces the synaptic levels of the scaffold proteins Dlg and α-spectrin. We also provide evidence that Atl functions with the microtubule-severing protein Spastin to disassemble microtubules in muscles. Finally, we demonstrate that the microtubule-destabilizing drug vinblastine alleviates synapse and muscle defects in <I>atl</I> mutants. Together, our results suggest that Atl controls synapse development and ER and Golgi morphogenesis by regulating microtubule stability.</P>
Tai Sun Park,You Na Oh,Sang-Bum Hong,Chae-Man Lim,Younsuck Koh,Je-Hwan Lee,Jung-Hee Lee,Kyoo-Hyung Lee,Jin Won Huh 대한중환자의학회 2016 Acute and Critical Care Vol.31 No.3
Background: Administering extracorporeal membrane oxygenation (ECMO) to critically ill patients with acute respiratory distress syndrome has substantially increased over the last decade, however administering ECMO to patients with hematologic malignancies may carry a particularly high risk. Here, we report the clinical outcomes of patients with hematologic malignancies and severe acute respiratory failure who were treated with ECMO. Methods: We performed a retrospective review of the medical records of patients with hematologic malignancies and severe acute respiratory failure who were treated with ECMO at the medical intensive care unit of a tertiary referral hospital between March 2010 and April 2015. Results: A total of 15 patients (9 men; median age 45 years) with hematologic malignancies and severe acute respiratory failure received ECMO therapy during the study period. The median values of the Acute Physiology and Chronic Health Evaluation II score, Murray Lung Injury Score, and Respiratory Extracorporeal Membrane Oxygenation Survival Prediction Score were 29, 3.3, and -2, respectively. Seven patients received venovenous ECMO, whereas 8 patients received venoarterial ECMO. The median ECMO duration was 2 days. Successful weaning of ECMO was achieved in 3 patients. Hemorrhage complications developed in 4 patients (1 pulmonary hemorrhage, 1 intracranial hemorrhage, and 2 cases of gastrointestinal bleeding). The longest period of patient survival was 59 days after ECMO initiation. No significant differences in survival were noted between venovenous and venoarterial ECMO groups (10.0 vs. 10.5 days; p = 0.56). Conclusions: Patients with hematologic malignancies and severe acute respiratory failure demonstrate poor outcomes after ECMO treatment. Careful and appropriate selection of candidates for ECMO in these patients is necessary.
Lee, Sukyee,Lee, Seung-Hun,VanBik, Dorene,Kim, Neung-Hee,Kim, Kyoo-Tae,Goo, Youn-Kyoung,Rhee, Man Hee,Kwon, Oh-Deog,Kwak, Dongmi Elsevier 2016 Ticks and tick-borne diseases Vol.7 No.5
<P><B>Abstract</B></P> <P>In this study, the status of <I>Anaplasma phagocytophilum</I> infection was assessed in shelter dogs in Seoul, Korea, with PCR and phylogenetic analyses. Nested PCR on 1058 collected blood samples revealed only one <I>A. phagocytophilum</I> positive sample (female, age <1year, mixed breed, collected from the north of the Han River). The genetic variability of <I>A. phagocytophilum</I> was evaluated by genotyping, using the 16S rRNA, <I>groEL</I>, and <I>msp2</I> gene sequences of the positive sample. BLASTn analysis revealed that the 16S rRNA, <I>groEL</I>, and <I>msp2</I> genes had 99.6%, 99.9%, and 100% identity with the following sequences deposited in GenBank: a cat 16S rRNA sequence from Korea (KR021166), a rat <I>groEL</I> sequence from Korea (KT220194), and a water deer <I>msp2</I> sequence from Korea (HM752099), respectively. Phylogenetic analyses classified the <I>groEL</I> gene into two distinct groups (serine and alanine), whereas the <I>msp2</I> gene showed a general classification into two groups (USA and Europe) that were further subgrouped according to region. To the best of our knowledge, this study is the first to describe the molecular diagnosis of <I>A</I>. <I>phagocytophilum</I> in dogs reared in Korea. In addition, the high genetic identity of the 16S rRNA and <I>groEL</I> sequences between humans and dogs from the same region suggests a possible epidemiological relation. Given the conditions of climate change, tick ecology, and recent incidence of human granulocytic anaplasmosis in Korea, the findings of this study underscore the need to establish appropriate control programs for tick-borne diseases in Korea.</P>