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ZICHUAN MA,YINSU WU,SHENGTAO XING,YONGFANG CHANG,XIAORU WEI 성균관대학교(자연과학캠퍼스) 성균나노과학기술원 2014 NANO Vol.9 No.7
Various manganese oxides nano/micro-crystals, including Mn 3 O 4 octahedrons, hierarchical °ow-er-like K- ? -MnO 2 microspheres, multi-branch and strip-shaped ? -MnOOH, have been prepared bya lignosulfanate (LSN)-mediated hydrothermal process. The e®ect of LSN on the structure andmorphology has been thoroughly investigated by X-ray powder di®raction and scanning electronmicroscopy (SEM). The results indicated that stepwise reduction and Ostwald's ripening occurredsimultaneously in this hydrothermal process, and LSN could serve as both reducing agent andgrowth modi¯er. The appropriate reducing capacity and selective absorption ability of LSN playedan important role for the formation of various manganese oxides nano/micro-crystals. The cat-alytic performance of the products for the oxidation of o-xylene has been also investigated. Theresult showed that the MnO 2 crystals prepared with LSN exhibited the highest catalytic activity.
Coal dust exposure induces proliferation and migration of human bronchial epithelial cells
Li Amin,Zhang Yinci,Wang Ruikai,Xu Ruyue,Ma Yongfang,Song Li,Cao Weiya,Xiaolong Tang 대한독성 유전단백체 학회 2023 Molecular & cellular toxicology Vol.19 No.2
Background Coal dust exposure has caused a variety of lung diseases. In addition to genotoxicity and cytotoxicity, other biological changes caused by coal dust (CD) exposure need further study. Objective To observe the cellular transformation eff ects of CD exposure and explore its underlying molecular mechanism, human bronchial epithelial cells (BEAS-2B) were cultured with continuous CD exposure. Methylthiazolyldiphenyl-tetrazolium bromide (MTT) assay, colony formation assay, wound healing assay, next-generation sequencing (NGS) and western blotting were performed to observe the cell proliferation, migration, genomic transcription and pathological signaling pathways. Results We demonstrated that BEAS-2B cells with long-term chronic CD exposure show accelerated proliferation rate and enhanced migration ability, and have altered gene expression profi les and aberrant activation of EGFR/Raf/ERK and PI3K/ AKT/mTOR pathways. Conclusions The results indicate that chronic CD exposure could induce abnormal proliferation and migration of BEAS-2B cells, lead to the transformation potential of human bronchial epithelial cells.