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- 저자 : Lu Shi-bao (검색결과 2 건)
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A Research of Engineering Construction Based on Credit Risk
Lu Shi-bao,Huang Qiang,ZHANG Yong-yong,SUN Dong-yong 인하대학교 정석물류통상연구원 2009 인하대학교 정석물류통상연구원 학술대회 Vol.2009 No.10
Engineering construction companies often default on the contract expird, which makes the enterprise"s own quality and credibility be affected. Therefore, the construction enterprises should be aware that the value of their own credit, financial sector should also be on the borrowers to assess credit risk, so as not to appear in the portfolio risk. This article utilizes the modern credit grading method toevaluate the construction enterprise"s Credit rating. The study found: Change index credit transpose matrix has a great impact. It will affect the credit rating chages. Credit Change this index ranged from zero when the critical state has a different default status, when the credit quality index greater than zero, the rise in the value of its credit; if the credit quality index is less than zero, the credit quality of the index on the lower Level; If zero, credit rating is tomaintain the normal status, and obtained the illustration through the example.
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Shan, Li-Na,Song, Yong-Gui,Su, Dan,Liu, Ya-Li,Shi, Xian-Bao,Lu, Si-Jing Asian Pacific Journal of Cancer Prevention 2015 Asian Pacific journal of cancer prevention Vol.16 No.9
The zinc finger transcription factor EGR 1 has a role in controlling synaptic plasticity, wound repair, female reproductive capacity, inflammation, growth control, apoptosis and tumor progression. Recent studies mainly focused on its role in growth control and apoptosis, however, little is known about its role in epithelial-mesenchymal transition (EMT). Here, we aim to explore whether EGR 1 is involved in TGF-${\beta}1$-induced EMT in non-smallcell lung cancer cells. Transforming growth factor (TGF)-${\beta}1$ was utilized to induce EMT in this study. Western blotting, RT-PCR, and transwell chambers were used to identify phenotype changes. Western blotting was also used to observe changes of the expression of EGR 1. The lentivirus-mediated EGR 1 vector was used to increase EGR 1 expression. We investigated the change of migration to evaluate the effect of EGR 1 on non-small-cell lung cancer cells migration by transwell chambers. After stimulating with TGF-${\beta}1$, almost all A549 cells and Luca 1 cells (Non-small-cell lung cancer primary cells) changed to mesenchymal phenotype and acquired more migration capabilities. These cells also had lower EGR 1 protein expression. Overexpression of EGR 1 gene with EGR 1 vector could decrease tumor cell migration capabilities significantly after adding TGF-${\beta}1$. These data s howed an important role of EGR 1 in the EMT of non-small-cell lung cancer cells, as well as migration.
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