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[PB-0007] Gene integration approach detected QTLs Associated with Bakanae disease resistance in rice
Sais-Beul Lee(Sais-Beul Lee),Hyunggon Mang(Hyunggon Mang),Ju-Won Kang(Ju-Won Kang),Ji-Yoon Lee(Ji-Yoon Lee),Nkulu Rolly Kabange(Nkulu Rolly Kabange ),Yeongho Kwon(Yeongho Kwon),Gi-Un Seong(Gi-Un Seong 한국육종학회 2022 한국육종학회 공동학술발표집 Vol.2022 No.-
[PB-0006] A novel resistance gene for bakanae disease in rice, qBK4T identified by GWAS
Sais-Beul Lee(Sais-Beul Lee),Ju-Won Kang(Ju-Won Kang),Ji-Yoon Lee(Ji-Yoon Lee),Nkulu Rolly Kabange(Nkulu Rolly Kabange ),Hyunggon Mang(Hyunggon Mang),Yeongho Kwon(Yeongho Kwon),Gi-Un Seong(Gi-Un Seong 한국육종학회 2022 한국육종학회 공동학술발표집 Vol.2022 No.-
Hye-Young Lee(이혜영),Ye-Eun Seo(서예은),Soohyun Oh(오수현),Hyunggon Mang(맹형곤),Doil Choi(최도일) 한국원예학회 2021 한국원예학회 학술발표요지 Vol.2021 No.10
Hypersensitive response (HR) is a robust immune response mediated by nucleotide-binding, leucine-rich repeat receptors (NLRs). However, the early molecular event that links activated NLRs to cell death is unclear. Here, we demonstrate that NLRs target plasma membrane H<SUP>+</SUP>-ATPases (PMAs) that generate electrochemical potential, an essential component of living cells, across the plasma membrane. CC<SUP>A</SUP>309, an autoactive N-terminal domain of a coiled-coil NLR (CNL), associates with PMAs. Silencing or overexpression of PMAs reversibly affects cell death induced by CC<SUP>A</SUP>309. CC<SUP>A</SUP>309-induced apoplast alkalization causes plasma membrane depolarization, followed by cell death. Co-immunoprecipitation analyses suggest that CC<SUP>A</SUP>309 inhibits PMA activation by pre-occupying the dephosphorylated penultimate threonine residue of PMA. Moreover, pharmacological experiments using fusicoccin, an irreversible PMA activator, showed that inhibition of PMAs contributes to CNL-type (but not TNL-type) resistance proteins-induced cell death. We suggest PMAs as primary targets of plasma membrane-associated CNLs leading to HR-associated cell death by disturbing the electrochemical gradient across the membrane. These results provide new insight into NLR-mediated cell death in plants.