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Myoclonus-Ataxia Syndrome Associated with COVID-19
Kuldeep Shetty,Atul Manchakrao Jadhav,Ranjith Jayanthakumar,Seema Jamwal,Tejaswini Shanubhogue,Mallepalli Prabhakar Reddy,Gopal Krishna Dash,Radhika Manohar,Vivek Jacob Philip,Vikram Huded 대한파킨슨병및이상운동질환학회 2021 Journal Of Movement Disorders Vol.14 No.2
Neurological manifestations of coronavirus disease (COVID-19) have increasingly been reported since the onset of the pandemic. Herein, we report a relatively new presentation. A patient in the convalescence period following a febrile illness with lower respiratorytract infection (fever, myalgia, nonproductive cough) presented with generalized disabling myoclonus, which is phenotypicallysuggestive of brainstem origin, along with additional truncal cerebellar ataxia. His neurology work-ups, such as brainMRI, electroencephalography, serum autoimmune and paraneoplastic antibody testing, were normal. His CT chest scan revealedright lower lung infiltrates, and serological and other laboratory testing did not show evidence of active infection. COVID-19 titers turned out to be strongly positive, suggestive of post-COVID-19 lung sequelae. He responded partially to antimyoclonicdrugs and fully to a course of steroids, suggesting a para- or postinfectious immune-mediated pathophysiology. Myoclonusataxiasyndrome appears to be a neurological manifestation of COVID-19 infection, and knowledge regarding this phenomenonshould be increased among clinicians for better patient care in a pandemic situation.