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최윤백 ( Y. B. Choi ),정훈용 ( H. Y. Jung ),김태원 ( T. W. Kim ),이영상 ( Y. S. Lee ),김명환 ( M. H. Kim ),김진호 ( J. H. Kim ),강윤구 ( Y. G. Kang ),서철원 ( C. W. Seo ) 대한소화기학회 2002 대한소화기학회 춘계학술대회 Vol.2002 No.-
<목적> 복강경의 이용증가와 복강경 수술기구의 급격한 발전에 힘입어 복강경은 복부질환의 진단 및 치료에 적극적으로 사용되고 있다. 특히 보편적인 검사 및 침습적이고 복잡한 고가의 검사에도 불구하고 진단이 확실히 않은 복부질환의 경우 개복술을 대신할 수 있는 진단방법으로서의 복강경의 안전성 및 의의를 알아보고자 본 연구를 시행하였다. <방법> 1999년 2월부터 2001년 5월까지 본원에서 진단목적으로 복강경처치가 시행된 67명의 환자를 대상으로 이들의
최윤백(Youn Baik Choi),육정환(Jeong Hwan Yook),이승규(Sung Gyu Lee) 대한소화기학회 1995 대한소화기학회지 Vol.27 No.3
Laparoscopic cholecystectomy has rapidly heen adopted by surgeons since 1987 and become the gold standard therapy for management of symptomatic gallstone disease, but concerns remain about its safety due to bile duct injury as compared with open cholecystectomy. As the complication of bile leaks due to bile duct injury can be devasting, its timely diagnosis and appropriate management are crucial. Especially in those patients whose clinically significant bile leaks do not resolve spontaneously, surgery is frequently employed for proper management. However, recent technical advances in interventional area(endoscopy and radioiogy) have fostered the successful management of bile leaks. Among 1548 cases of laparoscopic cholecystecto- mies, 3 cases of bile leaks are noticed. Endoscopic retrograde cholangiopancreaticography(ERCP) with endoscopic sphincterotomy(EST) proved to be a uset'ul diagnostic and therapeutic modality in the treatment of these patients. This report advocates the use of ERCP with EST and endobiliary stenting in the management of biliary injury resulting from laparoscopic cholecystectomy.(Korean J Gastroenterol 1995;375 - 380)
최윤백(Youn-Baik Choi),이종률(Jong-Youl Lee),곽진호(Jin-Ho Kwak),장혁재(Hyuk-Jai Jang),한명식(Myoung-Sik Han) 대한외과학회 2007 Annals of Surgical Treatment and Research(ASRT) Vol.72 No.5
Purpose: Inguino-femoral hernias in women are less common than that in a man, and we have had limited experience for hernia repair in women. The purpose of this study was to evaluate the characteristics of inguino-femoral hernias in females and to establish the choice of specific treatment for inguino-femoral hernia in females. Methods: This retrospective study was based on the medical records of 566 patients who underwent 657 cases of herniorrhaphies for treating inguino-femoral hernia in adult females from January 1998 to June 2006. We evaluated the incidence of hernia, the operative technique and the length of the operation, the complications and the postoperative recurrence rate. The operative findings and median time to reoperation for a recurrent hernia were also evaluated. Results: During the 8.5-year period, we performed 2,931 herniorrhaphies in 2,274 patients. Of these, 657 herniorrhaphies were done in females (22.4%). The types of hernia in females were indirect inguinal hernia (67.3%), direct inguinal hernia (10.2%), the pantaloon type (10%) and femoral hernia (14.9%). Femoral hernia was more frequent in females (14.9%) compared to males (3.5%) (P<0.001). The overall rate of reoperation due to incarceration in the females was higher (2.5%) than that in the men (1.1%)(P<0.001). Femoral hernias in females was found at reoperation in 39.7% compared with 17.2% in the males (P<0.001). Conclusion: The incidence of inguino-femoral hernia in females was higher than the results of most published studies and the reoperation rate was higher in females. The increased frequency of femoral hernia at reoperation in females suggests avoiding injuries to the posterior wall of the inguinal canal and the need for exploration of the femoral canal at the time of the primary operation.
최윤백(Choi, Youn-Baik),김기환(Kim, Ki-Whan) 대한생리학회 1986 대한생리학회지 Vol.20 No.1
The sufficient myoplasmic Ca<sup>++</sup> to react with the contractile proteins is necessary to induce contraction of a cardiac muscle. These Ca<sup>++</sup> for the production of muscle contraction are supplied from the three recognized Ca<sup>++</sup> sources; internal Ca<sup>++</sup> release via the sarcoplasmic reticulum(SR), Ca<sup>++</sup> influx through a gated Ca-channel in the membrane as a Isi, and Ca<sup>++</sup> transport by the mechanism of Na/ca exchange. However, it is still controversial which Ca<sup>++</sup> sources act as a main contributor for myoplasmic Ca<sup>++</sup>, Therefore, this study was undertaken in order to examine the Ca<sup>++</sup> sources for the contraction of frog ventricle. There is evidence that the SR is sparse in frog ventricular fibers, and that T-tubules are absent. Isolated ventricular strips of frog, Rana nigromaculata, were used in this experiment. Isometric tension was recorded by force transducer, and membrane potentials of ventricular muscles were measured through the intracellular glass microelectrodes, which were filled with 3M KCI and had resistance of 30±50MΩ. All experiments were performed at room temperature in a tris¡¤buffered Ringer solution which was aerated with 100% O<sup>2</sup>. Isotonic high K, low Na solution was used to induce K-contracture, K-contracture appeared at the concentration of 20 to 30mM-KCI and was potentiated in parallel with the increase in KCI concentration. The contracture had two components: an initial rapid phasic and a subsequent slow tonic contractile responses. Membrane Potentials measured at normal Ringer solution(2.5mM KCI) was -90 to -100 mV, and decreased linearly as the KCI concentration increased; -55mV at 20mM.KCI, -45mV at 30 mM.KCI, -30 mY at 50 mM.KCI, and -12 mV at 100 mM.KCI. K-contracture was evoked firstly at the membrane potential of -45 mV. The contracture was potentiated by the increase of bathing extracellular Ca<sup>++</sup> concentration. However, in the absence of Ca<sup>++</sup> the contracture was almost not induced by 50 mM.KCI solution. Caffeine(20mM) in normal Ringer solution, which is known to release Ca<sup>++</sup> from SR without substantial effects on the Ca<sup>++</sup> fluxes across the surface membrane, did not affect membrane potential and also not initiate contracture, but the caffeine in 20 mM-KCI Ringer solution produced a contracture. Above results suggest that the main Ca<sup>++</sup> source for the K¡¤contracture of frog ventricle is Ca<sup>++</sup> influx through the voltage-dependent Ca-channel, and that in the K-contracture at the concentration of 100 mM-KCI, the mechanism of Na/ca exchange also partly contributs, in addition to the Ca<sup>++</sup> influx.