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A Weighted Block-by-Block Decoding Algorithm for CPM-QC-LDPC Code Using Neural Network
( Zuohong Xu ),( Jiang Zhu ),( Zixuan Zhang ),( Qian Cheng ) 한국인터넷정보학회 2018 KSII Transactions on Internet and Information Syst Vol.12 No.8
As one of the most potential types of low-density parity-check (LDPC) codes, CPM-QC-LDPC code has considerable advantages but there still exist some limitations in practical application, for example, the existing decoding algorithm has a low convergence rate and a high decoding complexity. According to the structural property of this code, we propose a new method based on a CPM-RID decoding algorithm that decodes block-by-block with weights, which are obtained by neural network training. From the simulation results, we can conclude that our proposed method not only improves the bit error rate and frame error rate performance but also increases the convergence rate, when compared with the original CPM-RID decoding algorithm and scaled MSA algorithm.
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Longxue Cai,Xianfa Liu,Qicai Guo,Qi huang,Qiong Zhang,Zuohong Cao 한국유전학회 2020 Genes & Genomics Vol.42 No.1
Objective Aim of this study was to detect the expression of miR-15a in rats following chronic constriction injury (CCI) and to investigate the regulatory functions of miR-15a during neuropathic pain (NP) development. Methods CCI was performed in adult Sprague–Dawley rats to set up the rat model of neuropathic pain. MiR-15a agomir and scrambled control were delivered into the implanted catheter of rats. The mechanical allodynia and thermal hyperalgesia were assessed in both CCI- and sham-operated groups. Rat lumbar spinal cord tissues were harvested for mRNA and protein analyses. The primary spinal microglia were isolated from adult Sprague–Dawley rats and transfected with miR-15a mimics, scramble miRNA, miR-15a inhibitor or its corresponding negative control. Cell lysates were collected for mRNA and protein analyses. Results Compared to sham-operated group, the expression of miR-15a in CCI rats was significantly reduced, whereas neuroinflammation in spinal cord tissues was increased. Intrathecal administration of miR-15a agomir significantly attenuated CCI-induced NP and the levels of proinflammatory cytokines, including IL-6, IL-1β, and TNF-α. AKT3 was predicted and confirmed as a miR-15a-regulated gene. We further demonstrated that miR-15a overexpression downregulated the level of AKT3 in primary rat microglia and rat CCI model. Moreover, the upregulation of miR-15a induced the expressions of autophagy-associated proteins, suggesting that the regulation mechanism of miR-15a in NP development involves AKT3mediated autophagy via inhibiting the expression of AKT3. Conclusion Our findings indicated that miR-15a might serve as a promising therapeutic target for the management of NP through the stimulation of autophagic process.
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( Pan Long ),( Fengxia Fan ),( Bin Xu ),( Zhengmi He ),( Yuting Su ),( Ping Zhang ),( Jianwei Xie ),( Zuohong Chen ) 한국균학회 2020 Mycobiology Vol.48 No.3
Three hepatic failure poisoning incidents caused by Lepiota brunneoincarnata and Lepiota venenata mushrooms have been occurred in China in 2017, L. venenata has been described as a new species. However, the cyclopeptide toxins of these lethal mushrooms remain poorly understood. In this study, the composition and content of amatoxins in L. brunneoincarnata and L. venenata are analyzed and compared, the analysis of composition and content of amatoxins in L. venenata are reported for the first time. The results showed that b-amanitin (b-AMA), a-amanitin (a-AMA), amanin, and amaninamide were identified in L. brunneoincarnata, and a-AMA, amanin II (an analog of amanin), and an unknown compound were identified in L. venenata. The differences between L. brunneoincarnata and L. venenata in the identified compounds provide chemical evidence for L. venenata as a new species. Quantitative analysis shows that a-AMA concentrations in L. brunneoincarnata and L. venenata were 0.72-1.97mg/g dry weight, b-AMA concentrations in L. brunneoincarnata were 0.57-0.94mg/g dry weight, and b-AMA was absent in L. venenata.
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