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Self-Organization of Multi-UAVs for Improving QoE in Unequal User Distribution
Young Jeon,Wonseok Lee,Tran Manh Hoang,Taejoon Kim 한국인터넷정보학회 2022 KSII Transactions on Internet and Information Syst Vol.16 No.4
A self-organizing multiple unmanned aerial vehicles (multi-UAVs) deployment based on virtual forces has a difficulty in ensuring the quality-of-experience (QoE) of users because of the difference between the assumed center for users in a hotspot and an actual center for users in the hotspot. This discrepancy is aggravated in a non-uniform and mobile user distribution. To address this problem, we propose a new density based virtual force (D-VF) multi-UAVs deployment algorithm which employs a mean opinion score (MOS) as a metric of QoE. Because MOS is based on signal-to-noise ratio (SNR), a sum of users’ MOS is a good metric not only to secure a wide service area but to enhance the link quality between multi-UAVs and users. The proposed algorithm improves users’ QoE by combining virtual forces with a random search force for the exploration of finding multi-UAVs’ positions which maximize the sum of users’ MOS. In simulation results, the proposed deployment algorithm shows the convergence of the multi-UAVs into the position of maximizing MOS. Therefore, the proposed algorithm outperforms the conventional virtual force-based deployment scheme in terms of QoE for non-uniform user distribution scenarios.
Time Synchronization Robust to Topology Change Through Reference Node Re-Election
( Young Jeon ),( Taehong Kim ),( Taejoon Kim ),( Jaeseang Lee ),( Jae-hyun Ham ) 한국정보처리학회 2019 정보처리학회논문지. 컴퓨터 및 통신시스템 Vol.8 No.8
애드혹 네트워크에서는 하나의 기준노드를 선정하여 이를 중심으로 네트워크 내에 모든 노드들의 시간을 동기화하는 방법을 사용할 수 있다. 이러한 기준노드를 중심으로 하는 대표적인 시간동기화 알고리즘은 Flooding Time Synchronization Protocol (FTSP) 이다. 메시지를 주고 받는 과정에서 예측할 수 있는 지연과 예측할 수 없는 랜덤 지연이 발생하게 되는데, 이러한 지연은 정확한 동기화를 방해하기 때문에 제거해야 한다. 멀티홉 기반의 통신에서 메시지를 주고받는 과정에 지나는 홉 수에 따라 홉 지연이 발생하게 된다. 이러한 홉 지연은 노드 사이의 동기화 성능을 크게 떨어뜨리게 된다. 따라서 이러한 홉 지연을 줄이고 동기화 성능을 높이기 위한 방법이 필요하다. 기존 FTSP 방식에서 기준노드가 ID를 기반으로 가장 ID 값이 작은 노드가 기준노드로 선정되기 때문에 기준노드의 위치가 어디냐에 따라서 홉 지연에 따른 성능 저하가 크게 발생할 수 있다. 본 논문에서는 홉 지연을 줄이기 위한 최적의 기준노드 재선정 알고리즘을 제안하고 OPNET 네트워크 시뮬레이터를 사용하여 기존 FTSP와의 성능을 비교한다. 추가적으로 토폴로지 변경에 따른 성능을 측정하여 제안된 방식이 토폴로지 변환에 강인한 성능을 갖고 있음을 확인할 수 있었다. In an Ad-hoc network, a method of time synchronizing all the nodes in a network centering on one reference node can be used. A representative algorithm based on a reference node is Flooding Time Synchronization Protocol (FTSP). In the process of sending and receiving messages, predictable and unpredictable delays occur, which should be removed because it hinders accurate time synchronization. In multi-hop communications, hop delays occur when a packet traverses a number of hops. These hop delays significantly degrade the synchronization performance among nodes. Therefore, we need to find a method to reduce these hop delays and increase synchronization performance. In the FTSP scheme, hop delays can be greatly increased depending on the position of a reference node. In addition, in FTSP, a node with the smallest node ID is elected as a reference node, hence, the position of a reference node is actually arbitrarily determined. In this paper, we propose an optimal reference node election algorithm to reduce hop delays, and compare the performance of the proposed scheme with FTSP using the network simulator OPNET. In addition, we verify that the proposed scheme has an improved synchronization performance, which is robust to topology changes.
웨어러블 컴퓨터 환경의 개인 네비게이션 수행에 다중양식 단서가 미치는 영향
전하영(Ha?Young Jeon),채행석(Haeng?Suk Chae),홍지영(Ji?Young Hong),한광희(Kwang?Hee Han) 대한인간공학회 2007 大韓人間工學會誌 Vol.26 No.4
Navigation system or way finding in Wearable computer help disabled and impaired persons and it is impossible to be safe and efficient for drivers as well as pedestrian. Wearable computing situation must be multi-tasking simultaneously and users need minimal attention. In this paper, we used virtual environment as real way-finding similarly. The direction cues of navigation system are investigated as visual only, visual & auditory, and visual & speech. In the paper, the trial demonstrates the difference of performance in detection of directing and performance of motor and subjective satisfaction of user.
Prophylactic effects of swimming exercise on autophagy-induced muscle atrophy in diabetic rats
Youngjeon Lee,Joo-Heon Kim,Yunkyung Hong,Sang-Rae Lee,Kyu-Tae Chang,Yonggeun Hong 한국실험동물학회 2012 Laboratory Animal Research Vol.28 No.3
Diabetes decreases skeletal muscle mass and induces atrophy. However, the mechanisms by which hyperglycemia and insulin deficiency modify muscle mass are not well defined. In this study, we evaluated the effects of swimming exercise on muscle mass and intracellular protein degradation in diabetic rats, and proposed that autophagy inhibition induced by swimming exercise serves as a hypercatabolic mechanism in the skeletal muscles of diabetic rats, supporting a notion that swimming exercise could efficiently reverse the reduced skeletal muscle mass caused by diabetes. Adult male Sprague-Dawley rats were injected intraperitoneally with streptozotocin (60 mg/kg body weight) to induce diabetes and then submitted to 1 hr per day of forced swimming exercise, 5 days per week for 4 weeks. We conducted an intraperitoneal glucose tolerance test on the animals and measured body weight, skeletal muscle mass, and protein degradation and examined the level of autophagy in the isolated extensor digitorum longus, plantaris, and soleus muscles. Body weight and muscle tissue mass were higher in the exercising diabetic rats than in control diabetic rats that remained sedentary. Compared to control rats, exercising diabetic rats had lower blood glucose levels, increased intracellular contractile protein expression, and decreased autophagic protein expression. We conclude that swimming exercise improves muscle mass in diabetes-induced skeletal muscle atrophy, suggesting the activation of autophagy in diabetes contributes to muscle atrophy through hypercatabolic metabolism and that aerobic exercise, by suppressing autophagy, may modify or reverse skeletal muscle wasting in diabetic patients.
Autophagy contributes to retardation of cardiac growth in diabetic rats
Youngjeon Lee,Yunkyung Hong,Sang-Rae Lee,Kyu-Tae Chang,Yonggeun Hong 한국실험동물학회 2012 Laboratory Animal Research Vol.28 No.2
Diabetes mellitus is a major predictor of heart failure, although the mechanisms by which the disease causes cardiomyopathy are not well understood. The purpose of this study was to determine whether prolonged exposure of cardiomyocytes to high glucose concentrations induces autophagy and contributes to cardiomyopathy. Interestingly, there were no differences in the autophagic activation produced by different glucose concentrations. However, cell viability was decreased by high glucose. In the diabetic rats, we found a higher level of microtubule-associated protein light chain 3 (LC3) expression and a reduction in the size of the left ventricle (LV) (P<0.05) caused by growth retardation, suggesting activated autophagy. Our in vitro findings indicate that hyperglycemic oxidative stress induces autophagy, and our in vivo studies reveal that autophagy is involved in the progression of pathophysiological remodeling of the heart. Taken together, the studies suggest that autophagy may play a role in the pathogenesis of juvenile diabetic cardiomyopathy.
Therapeutically Targeting Neuroinflammation and Microglia after Acute Ischemic Stroke
Lee, Youngjeon,Lee, Sang-Rae,Choi, Sung S.,Yeo, Hyeon-Gu,Chang, Kyu-Tae,Lee, Hong J. Hindawi Publishing Corporation 2014 BioMed research international Vol.2014 No.-
<P>Inflammation has a pivotal role in the pathogenesis of ischemic stroke, and recent studies posit that inflammation acts as a double-edged sword, not only detrimentally augmenting secondary injury, but also potentially promoting recovery. An initial event of inflammation in ischemic stroke is the activation of microglia, leading to production of both pro- and anti-inflammatory mediators acting through multiple receptor signaling pathways. In this review, we discuss the role of microglial mediators in acute ischemic stroke and elaborate on preclinical and clinical studies focused on microglia in stroke models. Understanding how microglia can lead to both pro- and anti-inflammatory responses may be essential to implement therapeutic strategies using immunomodulatory interventions in ischemic stroke.</P>
Lee, Youngjeon,Lee, Seunghoon,Lee, Sang-Rae,Park, Kanghui,Hong, Yunkyung,Lee, Minkyung,Park, Sookyoung,Jin, Yunho,Chang, Kyu-Tae,Hong, Yonggeun Molecular Diversity Preservation International (MD 2014 INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES Vol.15 No.2
<P>Endogenous neural stem/progenitor cells (eNSPCs) proliferate and differentiate into neurons and glial cells after spinal cord injury (SCI). We have previously shown that melatonin (MT) plus exercise (Ex) had a synergistic effect on functional recovery after SCI. Thus, we hypothesized that combined therapy including melatonin and exercise might exert a beneficial effect on eNSPCs after SCI. Melatonin was administered twice a day and exercise was performed on a treadmill for 15 min, six days per week for 3 weeks after SCI. Immunohistochemistry and RT-PCR analysis were used to determine cell population for late response, in conjunction with histological examination and motor function test. There was marked improvement in hindlimb function in SCI+MT+Ex group at day 14 and 21 after injury, as documented by the reduced size of the spinal lesion and a higher density of dendritic spines and axons; such functional improvements were associated with increased numbers of BrdU-positive cells. Furthermore, MAP2 was increased in the injured thoracic segment, while GFAP was increased in the cervical segment, along with elevated numbers of BrdU-positive nestin-expressing eNSPCs in the SCI+MT+Ex group. The dendritic spine density was augmented markedly in SCI+MT and SCI+MT+Ex groups. These results suggest a synergistic effect of SCI+MT+Ex might create a microenvironment to facilitate proliferation of eNSPCs to effectively replace injured cells and to improve regeneration in SCI.</P>