http://chineseinput.net/에서 pinyin(병음)방식으로 중국어를 변환할 수 있습니다.
변환된 중국어를 복사하여 사용하시면 됩니다.
Three Mile Island: Medical and Public Health Aspects of a Radiation Accident
Linnemann,Roger E. 대한방사선 방어학회 1981 방사선방어학회지 Vol.6 No.1
The March 1979 accident at Three Mile Island provided physicians specializing in radiation medicine an opportunity to observe the field under conditions never seen before. Since no injuries occurred at the site or within the community, medical personnel were immediately involved in efforts to allay fear, provide accurate information, and replace laboratory resources rendered ineffective by the release in the reactor building. Valuable insights concerning medical emergency planning are derived from the accidents; suggestions are made for handling any future mishaps.
Initial Management of Radiation Injuries
Linnemann,Roger E. 대한방사선방어학회 1980 방사선방어학회지 Vol.5 No.1
The increasing utilization of radioactive isotopes in industry, medicine and research has raised the question, “How should hospitals deal with radiation injuries when they occur?” A system for initial management of radiation injuries has been developed by Radiation Management Corporation. Radiation injuries are classified and a treatment plan outlined for each at the emergency and short term medical care phase. This system includes clinical prognosis as well as a detailed alan for quick set up of a Radiation Emergency Area in any hospital. Procedures for patient admission, preparation of the facility, general decontamination, sample taking, and wound decontamination are included.
Inhibitory G proteins and their receptors: emerging therapeutic targets for obesity and diabetes
Michelle E Kimple,Joshua C Neuman,Amelia K Linnemann,Patrick J Casey 생화학분자생물학회 2014 Experimental and molecular medicine Vol.46 No.-
The worldwide prevalence of obesity is steadily increasing, nearly doubling between 1980 and 2008. Obesity is often associated with insulin resistance, a major risk factor for type 2 diabetes mellitus (T2DM): a costly chronic disease and serious public health problem. The underlying cause of T2DM is a failure of the beta cells of the pancreas to continue to produce enough insulin to counteract insulin resistance. Most current T2DM therapeutics do not prevent continued loss of insulin secretion capacity, and those that do have the potential to preserve beta cell mass and function are not effective in all patients. Therefore, developing new methods for preventing and treating obesity and T2DM is very timely and of great significance. Thereis now considerable literature demonstrating a link between inhibitory guanine nucleotide-binding protein (G protein) and G protein-coupled receptor (GPCR) signaling in insulin-responsive tissues and the pathogenesis of obesity and T2DM. These studies are suggesting new and emerging therapeutic targets for these conditions. In this review, we will discuss inhibitory G proteins and GPCRs that have primary actions in the beta cell and other peripheral sites as therapeutic targets for obesity and T2DM, improving satiety, insulin resistance and/or beta cell biology.