http://chineseinput.net/에서 pinyin(병음)방식으로 중국어를 변환할 수 있습니다.
변환된 중국어를 복사하여 사용하시면 됩니다.
웨이브렛 변환을 이용한 초음파 펄스 에코 신호의 디컨볼루션
장경영,장효성,박병일,하욥 한국비파괴검사학회 2000 한국비파괴검사학회지 Vol.20 No.6
초음파 펄스-에코법을 매우 얇은 층을 갖는 다층구조물에 적용할 때 그 얇은 층의 상하면에서의 반사파가 중첩되게 되면 검사가 곤란하게 된다. 이런 문제는 반도체 내부에서의 심각한 감쇠를 피하기 위해 20MHz 이하의 비교적 저주파수를 사용하는 초음파 현미경으로 반도체의 얇은 실리콘 칩을 검사하는 경우에 쉽게 볼 수 있다. 기존에 이런 초음파 신호의 중첩을 분리하기 위해 디컨볼루션 기법이 사용되어 왔으나, 송신파의 파형이 전파하면서 왜곡되어 수신되는 경우에는 적절치 못하다. 본 논문에서는 기존의 디컨볼루션 기법이 사용되어 왔으나, 송신파의 파형이 전파하면서 왜곡되어 분리해 낼 수 있는 새로운 신호처리 기법으로서 웨이브렛 변환 기반 디컨볼루션 (WTBD) 기법을 제안한였다. 여기서 웨이브렛 변환은 송신파와 왜곡된 수신 신호의 공통 파형을 추출하기 위해 사용되고 추출된 공통 파형에 대해 디컨볼루션 처리한다. 제안하는 방법의 성능은 모형신호에 대한 컴퓨터 시뮬레이션과 인위적으로 실리콘 칩 상면에 들뜸 결함을 만든 반도체 시편에 대한 실험을 통해 검증되었다. Ultrasonic pulse echo method comes to be difficult to apply to the multi-layered structure with very thin layer, because the echoes from the top and the bottom of the layer are superimposed. Wecan easily meet this problem when the silicon chip layer in the semiconductor is inspected by a SAM epquipment using fairly low frequency lower than 20MHz by which sebere attenuation in the epoxy mold compound of packaging material can be overcome. Conventionally, deconvolution technique has been used for the decomposirion of superromposed UR signal, however it has disavilities when the waveform pf the transmitted signals is distorted according th tje prpagation. In this paper, the wavelet transform based deconvolution (WTBD)technique is ptoposed as a new signal processing processing method that can decompose the superimpose the echo signals with superior performances compared to the conventional decovolution technique. WTBD method uses the wavelet transform in the pre-stage of deconvolution to extract out the common waveform from the transmitted and received signal with distortion. Performances of the proposed mehtod are shown by through computer simulations using model signal with noise and are demonstrated by through experiments for the fabricated semiconductor sample with partial delamination at the pot of silicon chip layer.
자매에서 발생한 중증 마이코플라즈마 폐렴에서 스테로이드 치료
김도희,이경일,김명숙,윤유숙,황자영,임정우,강진한,이준성 대한감염학회 2009 감염과 화학요법 Vol.41 No.3
Numerous evidences support the pathogenesis that M. pneumonias pneumonia is associated with cell-mediat-ed immune reaction. We report 2 cases of severe M. pneumoniae pneumonia in previously healthy sisters, who were both admitted during an epidemic of M. pneumoniae pneumonia. The elder sister, who was 16 years old, was admitted with M. pneumoniae pneumonia. She showed no response to treatment with clarithroycin and levo-floxacin, and eventually progressed to severe ARDS, requiring mechanical ventilation. After treatment with hydrocortisone (200 mg/day), there were rapid improvements in clinical manifestations and chest radiographic findings. The younger sister, who was 14 years old, was admitted 10 days later, Presenting with fever but no pneumonic lesions on chest radiograph. Just like her sister, the infection showed no response to clarithromycin. Fever persisted and pneumonic consolidation with mild pleural effusion was noticed in the left lower lobe on the 5th hospital-day. After treatment with oral prednisolone (1 mg/kg), she showed rapid defervescence and on the 8th hospital day, no pneumonic lesions were detectable on chest radiograph. Given the fact that the pathogenesis of pneumonia and extrapulmonary manifestations in M. pneumoniae infection is immune-mediated, an immuno-suppressive therapy would be validated for selected patients with M. pneumoniae infections.
( Kyung-yil Lee ) 대한신장학회 2017 Kidney Research and Clinical Practice Vol.36 No.2
Every cell of an organism is separated and protected by a cell membrane. It is proposed that harmony between intercellular communication and the health of an organism is controlled by a system, designated the proteinhomeostasis- system (PHS). Kidneys consist of a variety of types of renal cells, each with its own characteristic cellreceptor interactions and producing characteristic proteins. A functional union of these renal cells can be determined by various renal function tests, and harmonious intercellular communication is essential for the healthy state of the host. Injury to a kind of renal cells can impair renal function and induce an imbalance in total body health. Every acute or chronic renal disease has unknown etiologic substances that are responsible for renal cell injury at the molecular level. The immune/repair system of the host should control the etiologic substances acting against renal cells; if this system fails, the disease progresses to end stage renal disease. Each renal disease has its characteristic pathologic lesions where immune cells and immune proteins, such as immunoglobulins and complements, are infiltrated. These immune cells and immune proteins may control the etiologic substances involved in renal pathologic lesions. Also, genetic renal diseases and cancers may originate from a protein deficiency or malfunctioning protein under the PHS. A unified pathogenesis for renal diseases, including acute glomerulonephritis, idiopathic nephrotic syndrome, immunoglobulin A nephropathy, genetic renal diseases such as Alport syndrome, and malignancies such as Wilms tumor and renal cell carcinoma, is proposed using the PHS hypothesis.