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- 저자 : Barter (검색결과 4 건)
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A parking policy typology for clearer thinking on parking reform
Paul A. Barter 서울시립대학교 도시과학연구원 2015 도시과학국제저널 Vol.19 No.2
This paper contends that the absence of a widely understood typology of parking policy approaches is causing confusion in an important urban policy arena. This is apparent across the parking policy literature, both academic and practical, and across several regions. Previous typologies are reviewed and found to be either incomplete, overly simplistic, inaccurate, or failing to offer insight beyond merely describing the diversity. None enables much insight into the thinking behind each approach and reform thrust. To remedy this gap, a new approach to classifying parking policies is proposed. It is based on making explicit the contrasting mindsets behind different parking reform directions. A review of geographical diversity (both international and within metropolitan areas) is presented. This allows the value of the taxonomy to be evaluated, as well as enabling some refinements. Three main mindsets are posited, with each being defined by answers to two key questions. Each mindset has contrasting assumptions about the nature of parking as an economic good. Further detail in the typology is enabled through a third dimension based on one further question. New clarity provided by the new classification approach should reduce the tendency for parking debates to be confounded by the conflation of distinct reforms, by false dichotomies and by ‘straw man’ portrayals of key alternatives.
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High Density Lipoprotein: A Therapeutic Target in Type 2 Diabetes
Philip J. Barter 대한내분비학회 2013 Endocrinology and metabolism Vol.28 No.3
High density lipoproteins (HDLs) have a number of properties that have the potential to inhibit the development of atherosclerosis and thus reduce the risk of having a cardiovascular event. These protective effects of HDLs may be reduced in patients with type 2 diabetes, a condition in which the concentration of HDL cholesterol is frequently low. In addition to their potential cardioprotective properties, HDLs also increase the uptake of glucose by skeletal muscle and stimulate the synthesis and secretion of insulin from pancreatic β cells and may thus have a beneficial effect on glycemic control. This raises the possibility that a low HDL concentration in type 2 diabetes may contribute to a worsening of diabetic control. Thus, there is a double case for targeting HDLs in patients with type 2 diabetes: to reduce cardiovascular risk and also to improve glycemic control. Approaches to raising HDL levels include lifestyle factors such as weight reduction, increased physical activity and stopping smoking. There is an ongoing search for HDL-raising drugs as agents to use in patients with type 2 diabetes in whom the HDL level remains low despite lifestyle interventions.
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The Causes and Consequences of Low Levels of High Density Lipoproteins in Patients with Diabetes
Philip J. Barter 대한당뇨병학회 2011 Diabetes and Metabolism Journal Vol.35 No.2
Type 2 diabetes is commonly accompanied by a low level of high density lipoprotein cholesterol (HDL-C) that contributes to the increased cardiovascular risk associated with this condition. Given that HDLs have the ability to improve increase the uptake of glucose by skeletal muscle and to stimulate the secretion of insulin from pancreatic beta cells the possibility arises that a low HDL concentration in type 2 diabetes may also contribute to a worsening of diabetic control. Thus, there is a double case for raising the level of HDL.C in patients with type 2 diabetes: to reduce cardiovascular risk and to improve glycemic control. Approaches to raising HDL.C include lifestyle factors such as weight reduction, increased physical activity and stopping smoking. Of currently available drugs, the most effective is niacin. Newer formulations of niacin are reasonably well tolerated and have the ability to increase HDL.C by up to 30%. The effect of niacin on cardiovascular events in type 2 diabetes is currently being tested in a large-scale clinical outcome trial.
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