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Hot - Wall Epitaxy에 의한 CdTe(100) 박막의 성장과 광전류 특성
신현길(Hyun Keel Shin),신영진(Yeong Jin Shin),문종대(Jong Dae Moon) 한국진공학회(ASCT) 1994 Applied Science and Convergence Technology Vol.3 No.1
Hot-wall epitaxy 방법에 의하여 GaAs(100) 기판 위에 CdTe(100) 박막을 성장하였다. 박막을 성장하는 동안 기판의 온도는 280℃, 증발원의 온도는 430℃로 유지하였고 성장률은 2 ㎛/h이었다. 박막의 두께가 증가함에 따라 격자상수와 2결정 X-선 요동곡선의 반폭치가 감소하였다. CdTe(100) 박막의 광전류 스펙트럼으로부터 에너지 띠간격의 광전류 봉우리는 가전자대 Γ_8에 있는 전자가 광흡수에 의해 전도대 Γ_6로 전이한 것이며, 에너지 띠간격의 온도의존성을 구한 결과 온도계수 a=-2.3~-3.5×10^(-4) eV/K 정도임을 알았다. CdTe(100) epilayers were grown on GaAs(100) substrates by hot-wall epitaxy. During the growth, the substrate and source temperatures were maintained at 280℃ and 430℃, respectively. The growth rate of the epilayers was 2 ㎛/h. The lattice parameter and full width at half maximum of double crystal x-ray rocking curve were decreased as the epilayer thickness increased. From the photocurrent spectrum of the CdTe(100) epilayer the photocurrent peak corresponding to the band gap was thought to be due to the direct transition from the valence Γ_8 to the conduction Γ_6 with the absorption of light. From the variation of the band gap energy with temperature the temperature coefficients were about -2.3~-3.5×10^(-4)eV/K.
흡연과 낮은 방사선 피폭량이 Lymphocyte DNA 손상에 미치는 영향
신현길(Heuyn-Kil Shin),김윤주(Yun-Joo Kim),권은혜(Eun-Hye Kwon),육진영(Jin-Young Yook),최수용(Soo-Yong Choi) 환경독성보건학회 2003 환경독성보건학회지 Vol.18 No.4
Single cell gel electrophoresis (SCGE) was used to the experiment with the variation on the amount of smoking and low dose radiation exposure to find how much the Lymphocyte DNA was damaged, and especially for whom smoke a lot (about 20 or more than 20 cigarettes a day) it was found to be highly damaged. While, the damage of 'not more than 20 cigarettes a day' was found to be not so much significant as like for whom smoke about or more than 20 cigarettes a day. And, according to the different amount of the radiation exposure, the Lymphocyte DNA was found to be considerably damaged for 0~13 m Sv (P<0.01), it was not able to prove the relationship between the DNA damage and the radiation exposure.