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정원석 ( Won Seok Jung ), 서상완 ( Sang Wan Seo ), 조준기 ( Joon Ji Cho ), 손지우 ( Ji Woo Son ), 박민철 ( Min Cheol Park ), 최창민 ( Chang Min Choi ), 염승룡 ( Ryong Seung Yeom ), 황상욱 ( Sang Wook Hwang ), 김영우 ( Yong Woo Kim ), 송달) 대한본초학회 2006 大韓本草學會誌 Vol.21 No.3
Acute thromboembolic occlusion of the superior mesenteric artery (SMA) is a life-threatening condition with high mortality, and the lack of accurate and readily available diagnostic clues often results in delay in diagnosis and treatment. We describe the case of a 68-year-old man suffering from SMA thromboembolism presenting as acute heart failure.
Fibrous dysplasia very commonly involves the proximal femur. Fibrous dysplasia of the proximal end of the femur is a serious clinical problem because structural stabilty is destroyed and function of the hip joint is impaired due to the pathologic fracture and angulation deformity which influenced by biomechanical structure of the femur. So, the stability and functional reconstruction of the hip joint must considered at the treatment of the fibrous dysplasia of the proximal end of the femur. Authors have reviewed a series of 14 patients of fibrous dysplasia which were treated at the Department of Orthopedic Surgery, School of Medicine, Kyung Hee University. The follow-up period ranged from January 1981 to December 1990, the average being 3 years and 6 months. The average age was 21.8 years. Among 14 patients, the nine patients were monostotic type and the five patients were polyostotic type. And one polyostotic type showed bilateral involvement. The characteristics of the fibrous dysplasia around the hip joint were pain of the hip, coxa vara, angulation deformity, pathologic fracture of the femur was not delayed. Ten patients (7196) revealed satisfactory result, and four patients marked unsatisfactory result by the Stephensons classification. Simple curettage and bone graft couldnt prevent the progression of deformity or pathologic fracture in widely expanded lesion. The pro-phylactic rigid internal fixation with massive bone graft was mandatory to prevent the progression of deformity or pathologic fracture.