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난소와 자궁내경부에 동시에 발생한 원발성 악성종양 1예
권성택 ( Sung Taeg Kwon ),김진성 ( Jin Sung Kim ),최원준 ( Won Joun Choi ),정영택 ( Young Taeg Joung ),이순애 ( Soon Ae Lee ),이종학 ( Jong Hak Lee ),김종화 ( Jong Hwa Kim ),고경혁 ( Kyoung Hyuck Ko ),백원영 ( Won Young Paik ) 대한산부인과학회 1997 Obstetrics & Gynecology Science Vol.40 No.12
Synchronous primary neoplasms are as neoplasms diagnosed simultaneously or within a 6 month interval. The synchronous occurrence of carcinoma confined to the ovary and endocervix has been regarded as a very rare entity and presents a diagnostic and therapeutic dilemma. Recently we experienced 38 year old female patient who had mucinous cystadenocarcinoma of bilateral ovaries and adenocarcinoma in the uterine cervix simultaneously. So, we present the case with a review of the literatures.
Helicobacter pylori의 Kato III 세포내 발견
백승철,이정희,고경혁,조명제,조중현,김유경,윤희상,이우곤,이광호,강수민 대한소화기학회 1998 대한소화기학회지 Vol.31 No.6
Background/Aims; Some investigaters reported that the invasion of H. pylori into the gastric epithelial cells or lamina propria had been detected by light microscopie examination using various staining methods (i,e. the Warthin-Starry stain, immunohistologic stain) or by electron microscopic examination. However, the results are hardly convinced because they may be nonspecifically stained, or the intracytoplasmic organelles of the gastric epithelial cells may be mistaken for bacteria. Thus, we combined an immunohistologic method with an electron microscopy to deterrnine whether H. pylori is invasive into the gastric epithelial cells. Metbods: To enhance bacterial invasion, we adapted an in vitro system using Kato III cells, a gastric carcinoma eell line. The cells were infected with H. pylori. The infection was confirmed by staining with polyelonal antibody against H. pylori. and an electron microscopy. Resnlts: H. pylori was found within the cytoplasm of several Kato III cells. The intracellular bacteria could be differentiated from intracellular organelles or other bacteria-looking materials by gold particles attached to the bacteria, Conclusions: We could not elucidate the invasiveness of H. pylori, but we found the presence of H. pylori in the cytoplasm of the Kato III cells. These results indicate that the invasiveness of H. pylori will remain as one of the factors participating in the pathogenesis oF H. pylori-induced chronic gastritis and peptic ulcer. Further studies will be needed to demonstrate the invasiveness of H. pylori.