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Canine susceptibility to human influenza viruses (A/pdm 09H1N1, A/H3N2 and B)
Song, Daesub,Kim, Hyekwon,Na, Woonsung,Hong, Minki,Park, Seong-Jun,Moon, Hyoungjoon,Kang, Bokyu,Lyoo, Kwang-Soo,Yeom, Minjoo,Jeong, Dae Gwin,An, Dong-Jun,Kim, Jeong-Ki Society for General Microbiology 2015 The Journal of general virology Vol.96 No.2
<P>We investigated the infectivity and transmissibility of the human seasonal H3N2, pandemic (pdm) H1N1 (2009) and B influenza viruses in dogs. Dogs inoculated with human seasonal H3N2 and pdm H1N1 influenza viruses exhibited nasal shedding and were seroconverted against the viruses; this did not occur in the influenza B virus-inoculated dogs. Transmission of human H3N2 virus between dogs was demonstrated by observing nasal shedding and seroconversion in naïve dogs after contact with inoculated dogs. The seroprevalence study offered evidence of human H3N2 infection occurring in dogs since 2008. Furthermore, serological evidence of pdm H1N1 influenza virus infection alone and in combination with canine H3N2 virus was found in the serum samples collected from field dogs during 2010 and 2011. Our results suggest that dogs may be hosts for human seasonal H3N2 and pdm H1N1 influenza viruses.</P>
NIHSS 활용을 통한 뇌졸중 환자 간호의 질향상 전략
박경현,송경애,공혜성,나화주,유수경,박소영,김상희,최혜림,배희준,한문구,양미화 한국의료QA학회 2009 한국의료질향상학회지 Vol.15 No.2
문제: 뇌졸중 환자를 돌보는 간호현장에서는 급성기 뇌졸중 환자의 증상악화에 대한 조기사정과 적절한 대처가 매우 중요한 문제이다. 그러나 임상현장에서는 이를 객관화하고 수치화하여 모든 의료진이 의사소통할 수 있는 유용한 도구를 사용하고 있지는 못하는 실정이다. 이러한 문제를 해결하기위해 NIHSS(National Institute of Health Stroke Scale)를 신경과 진료의사를 중심으로활용하고 있지만 아직 간호현장에서는보편적으로사용하지 않고있다. 이에 뇌졸중환자간호에 NIHSS를 활용하여 뇌졸중 증상악화의 조기사정과 빠른 대처로 궁극적으로는 뇌졸중환자 간호의 질을보다 향상시키기 위하여 본활동이 시도되었다. 목적: 첫째, 간호사의 NIHSS 측정 신뢰도의 현수준을 점검하고 이를향상시키기 위한 프로그램을 개발·적용한다. 둘째, 간호사가급성기 뇌졸중환자에게 NIHSS 측정을 통하여 뇌졸중악화를 얼마나 예측할 수 있는지 분석하고 이를 향상시킬수있는 방법을 모색한다. 셋째, 궁극적으로 급성기 뇌졸중 환자의 증상악화의 조기 발견과치료를위한의료진의 올바른 대처지침을 마련하여 이를 적용한다. 의료기관: 경기도에 소재한 대학병원의 뇌졸중 집중치료실 질 향상 활동: 첫째, 간호사의 NIHSS 측정 신뢰도 향상을 위해 「NIHSS 간호사 교육 프로그램」을 마련하였다.
Diagnostic Tools of Pleural Effusion
Na, Moon Jun The Korean Academy of Tuberculosis and Respiratory 2014 Tuberculosis and Respiratory Diseases Vol.76 No.5
Pleural effusion is not a rare disease in Korea. The diagnosis of pleural effusion is very difficult, even though the patients often complain of typical symptoms indicating of pleural diseases. Pleural effusion is characterized by the pleural cavity filled with transudative or exudative pleural fluids, and it is developed by various etiologies. The presence of pleural effusion can be confirmed by radiological studies including simple chest radiography, ultrasonography, or computed tomography. Identifying the causes of pleural effusions by pleural fluid analysis is essential for proper treatments. This review article provides information on the diagnostic approaches of pleural effusions and further suggested ways to confirm their various etiologies, by using the most recent journals for references.
Diagnostic Tools of Pleural Effusion
Moon Jun Na 대한결핵 및 호흡기학회 2014 Tuberculosis and Respiratory Diseases Vol.76 No.5
Pleural effusion is not a rare disease in Korea. The diagnosis of pleural effusion is very difficult, even though the patients often complain of typical symptoms indicating of pleural diseases. Pleural effusion is characterized by the pleural cavity filled with transudative or exudative pleural fluids, and it is developed by various etiologies. The presence of pleural effusion can be confirmed by radiological studies including simple chest radiography, ultrasonography, or computed tomography. Identifying the causes of pleural effusions by pleural fluid analysis is essential for proper treatments. This review article provides information on the diagnostic approaches of pleural effusions and further suggested ways to confirm their various etiologies, by using the most recent journals for references.
기관지 천식환자에서 단기간 Prednisolone 경규투여후 나타난 기관지 과반응성 및 면역학적 지표의 변화
나문준(Moon Jun Na),이선우(Sun Woo Lee),신인철(In Cheol Shin),김영준(Young Jun Kim),박인원(In Won Park),최병휘(Byung Whui Choi),허성호(Sung Ho Hue) 대한내과학회 1992 대한내과학회지 Vol.43 No.2
N/A Recent studies have suggested that inflammation may play an important role in the characteristic bronchial hyperresponsivenees (BHR) and symptoms of bronchial asthma. The corticosteroid is widely used in bronchial asthmatics with antiinflammatory action, and improves the late response or bronchial hyperreactivity by the effect on the airway smooth muscle. Also, corticosteroid alters the immunologic properties by the mechanism which effects on the blood cells and their chemical mediators. In order to assess the effect of oral short-term prednisolone therapy on BHR in mild bronchial asthmatics, we measured nonspecific-BHR with methacholine bronchial provocation test [Meth-PC20 (mg/ml)], including IgE and eosinopohil count in peripheral blood in 2S bronchial asthmatic patients before and after treatment. Thery were divided into two groups; the control group (n=14) was treated with ordinary bronchodilator and antihistamine, and the steroid group (n=14) was treated with above regimen plus oral prednisolone (total 12wks; 30-40 mg/day for 1-2wks and tapering for 10-11wks). The results were as follows 1) Baseline FEV1 demonstrated no significant difference in both groups as 2340.00±500.56ml, 2296.43±514.29ml in control group, and 2387.14±683.48ml, 2474.29±660.74ml in steroid group before and after treatment. 2) Meth-PC20 was 8.92×/÷0.37 mg/ml before treatment and 12.29×/÷0.47 mg/ml after treatment in control group, which was not significantly changed. But after steroid therapy, Meth-PC showed significant change as 7.81 0.49 mg/ml vs 18.11×/÷0.51 mg/ml (p<0.05). 3) Peripheral eosinophil count was 272.25×/÷0.39/mm' before treatment and 254.38×/÷0.34/mm' after treatment in control group (p>0,05). In steroid group, it was 485. 83×/÷0.23/mm before treatment and 300.25×/÷0,40/mm after treatment (p0.05). 4) Total serum IgE measured as 147.38×/÷0.54 U/ml before treatment and 88.25×/÷0.43 U/ml after treatment in control group (p>0. 05). In steroid group, it was 563. 50×/÷0.64 U/ml before treatment and 302.92×/÷0.58U/ml after treatment (p<0.05). In conclusion, the oral corticosteroid therapy may reduce BHR in bronchial asthmatics with decrement of recruitment of activation of inflammatory cell, and inhibition of IgE production by the antiinflammatory effects.