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( Dong Chan Moon ),( Byung-yong Kim ),( Hyang-mi Nam ),( Geum-chan Jang ),( Suk-chan Jung ),( Hee-soo Lee ),( Yong-ho Park ),( Suk-kyung Lim ) 한국미생물 · 생명공학회 2016 Journal of microbiology and biotechnology Vol.26 No.4
Methicillin-resistant Staphylococcus aureus (MRSA), the major causative agent of nosocomial infection, has also been reported from non-human sources. A sequence type (ST) 541 MRSA isolate designated K12PJN53 was isolated from a healthy pig in 2012. The genome of K12PJN53 consists of 44 contiguous sequences (contigs), totalling 2,880,108 bases with 32.88% GC content. Among the annotated contigs, 14, 17, and 18 contained genes related to antimicrobial resistance, adherence, and toxin genes, respectively. The genomic distance of strain K12PJN53 was close to the ST398 strains. This is the first report of the draft genome sequence of a novel livestock-associated MRSA ST541 strain.
Molecular typing of Prototheca zopfii isolated from bovine mastitic milk samples in Korea
Ho Sung Park(Ho Sung Park),Su Jeong Kim(Su Jeong Kim),Geum-Chan Jang(Geum-Chan Jang),Bang-Hun Hyun(Bang-Hun Hyun),Suk-Kyung Lim(Suk-Kyung Lim),Dong Chan Moon(Dong Chan Moon) 한국예방수의학회 2017 한국예방수의학회 학술대회자료집 Vol.2017 No.-
Gwak, Geum-Youn,Yoon, Jung-Hwan,Yu, Su Jong,Park, Su Cheol,Jang, Ja June,Lee, Kyoung Bun,Lee, Sung-Hee,Lee, Soo-Mi,Shin, Chan Soo,Suh, Kyung-Suk,Lee, Hyo-Suk National Hellenic Research Foundation 2006 Oncology reports Vol.15 No.5
<P>N-cadherin signaling has recently been implicated in the progression of certain epithelial tumors by promoting invasion and dissemination of cancer cells. N-cadherin has also been reported to exert an anti-apoptotic effect. In this study, we attempted to evaluate the participation of this adhesion molecule in the progression of human hepatocellular carcinomas (HCCs) by analyzing its anti-apoptotic signaling as well as its prognostic implication in HCC patients. N-cadherin was found to be expressed in human HCCs. We established a stable human HCC cell line expressing a truncated N-cadherin, NCaddeltaC, with a dominant-negative action. NCaddeltaC-expressing cells were more susceptible to bile acid-induced apoptosis than control cells. N-cadherin was found to complex with procaspase-8, and this association was diminished in NCaddeltaC-expressing cells, leading to enhanced procaspase-8 recruitment to death-inducing signaling complex following bile acid treatment. A clinicopathological analysis in patients who had undergone surgical resection for HCC revealed that tumoral N-cadherin up-regulation was significantly related to poor recurrence-free and overall survival. Our findings implicate N-cadherin signaling as contributing to HCC progression by exerting anti-apoptotic effects. Thus, we suggest that the selective interruption of this signaling may have therapeutic potential.</P>