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精神分裂症으로 移行한 Gilles de la Tourette氏 病의 1例 報告
황익근 中央醫學社 1975 中央醫學 Vol.29 No.6
A case of Gilles de la Tourette's disease transformed to schizophrenic psychosis is reported in this paper. The patient has been suffered from Gilles de la Tourette's disease for 17 years without any reasonable treatment. He has not any history of Rheumatic fever or Encephalitis in his earlier life. E. E. G. , Skull X-Ray, and other neurological examinations do not reveal any abnormalities. The disease seems to be psychogenic in its origin in the light of the free neurological findings. During the course of his illness lasting 17 years, his personality has become gradually changed and destroyed resulting in schizophrenic psychoses. The anther also suggested the psychodynamic formulation about the cause of schizo?phrenic personality change and Gilles de la Tourette's disease in this case. It seems to be ambiguous that the Gilles de la Tourette's disease has really become transformed to schizophrenic psychoses or they have different origins in their process progressing respectively.
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Mescaline에 依한 Clonidine 效果의 中樞性 抉抗
黃翼根 전북대학교 의과학연구소 1980 全北醫大論文集 Vol.4 No.1
1. Antagonism of the intraventricular clonidine effect on blood pressure and heart rate by intraventricular mescaline was investigated in urethane-anesthetized rabbits. 2. Mescaline produced little effect on blood pressure, and produced slight decrease in heart rate. Clonidine produced distinct depressor and bradycardiac effects. 3. After mescaline administration, the depressor and bradycardiac effects of clonidine were markedly diminished and not observed, respectively. 4. The lowered blood pressure and the slowed heart rate by clonidine were heightened and temporalily increased by mescaline, respectively. 5. Intraventricular regitine did not affect the blood pressure response to mescaline but po-tentiated the decrease in heart rate by mescaline. 6. Methylatropine did not affect the decrease in heart rate by mescaline. 7. The results indicate that mescaline acts as an antagonist for the central α-adrenergic receptors. In addition mescaline may exert pulse-slowing effect of central origin
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黃翼根 전북대학교 의과학연구소 1981 全北醫大論文集 Vol.5 No.1
Clinical study was done for the child psychiatric patients who had been treated in the department of psychiatry, Jeonbug Medical School Hospital from 1975 to 1980. The results were as followings; 1. 14 percent of total psychiatric patients consisted of child psychiatric patients 2. Absolute number of child psychiatric patients has increased every year. 3. 75.2 percent of the total psychiatric patients belong to the age group between 12 and 16 years old. 4. Sex ratio for male and female was 1.6:1 5. 22.9 percent of the out-patients was neurosis, which was the highest among all of the disorders. 6. Neurosis(56.3%) psychosis(28.8%) and epilepsy(7.5%) were the major diseases in admitted patients. 7. 88.8 percent of the admitted patients belong to the puberty and early adolescent age group, and few patients were admitted under the age of 6.
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黃翼根 전북대학교 의과학연구소 1977 全北醫大論文集 Vol.1 No.-
An interesting case of bone marrow failure, which was caused by tridione, one of anti-epileptic drugs, has been reported in this paper. The changes of cellular elements in bone marrow and peripheral blood in relation to the toxicity of tridione on hematopoietic system have been reviewed briefly. Additionally, the effectiveness of combined therapy of testosterone and steroid to the case of bone marrow failure caused by tridione has been implied, and the clinical importance of regular blood examination to whom tridione is given has also been suggested.
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측뇌실내 Desmethylimipramine의 가토혈압 하강작용
황익근 大韓神經精神醫學會 1985 신경정신의학 Vol.24 No.3
1)Urethane 마취 가토에서, 측뇌실내 DMI(300㎍,1㎎)는 혈압 하강 반응을 일으켰고 정맥내 DMI(150,500㎍/㎏)는 뚜렷한 반응을 일으키지 않았다. 2)이 DMI의 혈압 하강 작용은, 측뇌실내 yohimb-ine, piperoxan으로 억제되었고, prazosin의 영향은 거의 받지 않았다. 3)이 DMI의 혈압 하강 작용은 측뇌실내 clonidine-저혈압상태에서는 거의 볼수 없었고, 측뇌실내 cl-onidine의 혈압 하강작용도 DMI-저혈압상태에서는 약화되었다. 4)Reserpine 처리가트에서는 측뇌실내 DMI(300㎍,1㎎)는 혈압상승을 일으켰으며, 이는 측뇌실내 yohimbine의 영향은 받지 않았으나 prazosin 투여후에는 거의 나타나지 않았다. 5)측뇌실내 Norepinephrine은 측뇌실내 DMI 300㎍ 후에는 혈압상승을 일으키지 않았으나 1㎎ 후에는 현저한 상승을 일으켰다. 6)측뇌실내 protriptyline은 혈압하강을 일으키지 않았다. 7)측뇌실내 DMI는 가토 뇌내 α₂-adrenoceptor에 작용하여 혈압 하강을 일으키며, 이는 DMI의 norepinephrine의 uptake 억제 효과에는 무관한 것으로 추측하였다. 1) Intraventricular DMI(300㎍,1㎎) produced a hypotensive response in urethane-anesthetized rabbits, howeber Intravenous DMI produced no distinctive effect. 2) The hypotensive effect of DMI was inhibited by treating rabbits with intraventricular yohimbi-ne or piperoxan, whereas intaventricular prazosin did not affect the effect. 3) The hypotensive effect of DMI was not pro-duced in clonidine-hypotension rabbits and the hypotensive effect of olonidine was weakened in DMI-hypotension rabbits. 4) In reserpine pretreated rabbits intraventricu-lar DMI(300㎍,1㎎) produced a hypertensive respones, which was not affected by treating the rabbits with yohimbine but was inhibited by pra-zosin. 5) Intraventricular norepinephrine produced lit-tle pressor effect in the rabbits pretreated with 300㎍ of intraventricular DMI, but it produced marked pressor effect after 1㎎ of intraventricu-lar DMI. 6) Intraventricular protriptyline did not cause hypotension. 7) It is inferred that intraventricular DMI pro-duced a hypotensive response in rabbits by acting on α₂-adrenoceptor, and this was not associated with the inhibitory effect of DMI on norepineph-rine uptake in adrenergic neurons.
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백서뇌의 Dopamine-β-hydroxylase의 성상에 관한 연구
황익근 전북대학교 의과학연구소 1982 全北醫大論文集 Vol.6 No.1
An experimental study was done in vitro about the nature of dopamine-beta-hydroxylase in mouse brain. The results are as following. 1. Brain dopamine-beta-hydroxylase activity of around 0.03n mol/mg protein/hr during 10 days after birth was similar to those of kinney, liver and intestine. The activity was similar to that of kidney in mature mouse. 2. Brain dopamine-beta-hydroxylase gradually increased during development with most remarkable change between 20 days after birth. Similar increased patern was observed in kidney. The induction of dopamine-bate-hydroxylase by thyroxine was most remarkable 10 days after birth and not observed 30 days after birth. 3. Diurnal variation of serum dopamine-beta-hydroxylase was found in mouse and the activity high around 11 p.m and low around 11 a.m.
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