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지방자치단체의 지역기록화 전략 수립을 위한 수정델파이 연구
엄상미 경인행정학회 2024 한국정책연구 Vol.24 No.1
본 연구는 지방자치단체의 지역기록화 전략 수립에 우선적으로 고려되어야 할 방안들을 제시하고자 하였다. 이를 위해 청주시 등 5개 지방자치단체의 사례를 분석하여 보편적 요인들을 추출하였고, 이를 기반으로 27명의 전문가 패널을 대상으로 수정델파이조사를 실시하였다. 그 결과 지역기록화의 수행 목적과 수행체계, 과업, 촉진 요인, 지역기록화의 효과 등 5개 영역 35개 항목에 대한 중요도를 파악하였다. 또한 지역기록화에 대한 지방자치단체의 책임성을 명확히 하였고, 책임수행을 위한 지방자치단체의 역할과 추진 방안들을 제시하였다. 본 연구는 지역기록화에 대한 표준화된 매뉴얼이 부재한 상황에서 기록전문가들의 경험과 지식에 기반하여 통합적, 다층적 탐색을 시도하였다는 점에 의의가 있다. 추후 지방자치단체별로 지역기록화 전략을 수립할 때 기준점을 제시할 수 있을 것으로 기대한다. This study aimed to propose alternative approaches that should be prioritized in formulating a locality documentation strategy for local governments. To achieve this, we conducted an analysis of cases from five local governments, including Cheongju City, and extracted universal factors based on them. We then conducted a Modified Delphi survey. As a result, we identified the importance of 35 items across five areas, including the purpose and system of regional record localization, tasks, promoting factors, and the effects of regional record localization. Additionally, we clarified the responsibility of local governments for regional record localization and presented their roles and implementation strategies for fulfilling this responsibility. This study is significant in that it attempted an integrated and multilayered exploration based on the experiences and knowledge of record experts, considering the absence of standardized manuals for regional record localization. It is expected that this study can provide reference points for formulating regional record localization strategies for each local government in the future.
엄상미,윤홍태,김용호 순천향대학교 기초과학연구소 2003 순천향자연과학연구 논문집 Vol.9 No.2
Phomopsis seed decay (PSD) is a major fungal disease found in most soybean production areas of the world. PSD significantly reduces soybean yield and seed quailty, particularly when soybean plants are exposed to high humidity and high temperature during maturation. To certificate genetic segregation ratio for PSD, PI 417479 line(resistant) and Hwaeomputkong(susceptible) were crossed and examined their inheritance mode. Experiment was conducted at glasshouse of Soonchunhyang Univ., in Korea. Different PSD seed infection levels were induced in soybean seeds by artificially infected plants exposing to above 95% relative humidity and 30˚C. A sample of seeds harvested from inoculated plants was analyzed to determine percent PSD incidence. The patterns of resistant response between Hwaeomputkong and PI 417479 were different in their seed and pod shape. The F₁plants of the resistance is governed by the two dominant genes. This results indicate that a resistance to PSD is controlled by complementary genes.
First report for Platanthera brevicalcarata (Orchidaceae) in Korea
엄상미,이남숙 한국식물분류학회 2012 식물 분류학회지 Vol.42 No.3
Platanthera brevicalcarata Hayata (Orchidaceae), previously known to be distributed from Taiwan to southern Japan, was found in the Jeju-do, Korea. This species is distinguished from the related taxon Platanthera chlorantha by its small plant size, white sepal with distinct one green vein, and short-cylindrical spur. The local name Young-ju-je-bi-ran is originated from the local name of Mt. Halla. The morphological characters and illustration of the species are provided with line drawing and photograph from the natural habitat.
Quantitative Transmission Disequilibrium Test를 이용한 지질대사관련 유전자의 혈중 지질농도 결정에 미치는 영향분석
박찬미,엄상미,장양수,박현영 대한심장학회 2008 Korean Circulation Journal Vol.38 No.4
Background and Objectives: The plasma lipid levels play crucial roles in the development of atherosclerotic diseases. We estimated the genetic variance of the lipid levels according to the contributions of the single nucleotide polymorphisms (SNPs) and haplotypes in 5 candidate genes. Subjects and Methods: We selected SNPs in the ATP binding cassette A1 (ABCA1) gene, the apolipoprotien A5 (APOA5), apolipoprotien E (APOE) gene, the cholesterol ester transfer protein (CETP) gene and the hepatic triglyceride lipase (LIPC) gene in 383 individuals from 100 Korean families. The genotype was determined by Orchid’s SNP-ITTM technology. The association analysis of the quantitative traits was performed using the quantitative transmission disequilibrium test. Results: A component analysis of the phenotypic variance explained 24.7% of the genetic variance on the total cholesterol, 26.4% of the genetic variance of the high density lipoprotein (HDL)-cholesterol, 11% of the genetic variance of the triglycerides, 35.6% of the genetic variance of the low density lipoprotein (LDL)-cholesterol and 18.9% of the genetic variance of the LDL-C/HDL-C, respectively. The association of the SNPs in the candidate genes explained a major fraction of the genetic phenotypic variance in the LDL-C/HDL-C ratio, but not in the other lipid profiles. The association with SNPs explained 38.5% of the variance for the total cholesterol, 32.2% of the variance for HDLcholesterol and 29.5% of the variance for LDL-cholesterol relative to the polygenic background. An analysis of the contribution of each gene to the genetic variance showed that ABCA1, APOE, CETP and LIPC influenced the variations in total cholesterol, LDL-cholesterol and LDL-C/HDL-C. The variation in HDL-cholesterol was influenced by ABCA1, APOA5 and APOE. Conclusion: We identified that the genetic variance for the total cholesterol, HDL-cholesterol and LDL-cholesterol, and the LDL-C/HDL-C ratio was significantly influenced by the genetic polymorphisms in 5 candidate genes in the Korean population. Further studies are necessary to identify other genes that can explain a major fraction of the genetic variance for the lipid levels. Background and Objectives: The plasma lipid levels play crucial roles in the development of atherosclerotic diseases. We estimated the genetic variance of the lipid levels according to the contributions of the single nucleotide polymorphisms (SNPs) and haplotypes in 5 candidate genes. Subjects and Methods: We selected SNPs in the ATP binding cassette A1 (ABCA1) gene, the apolipoprotien A5 (APOA5), apolipoprotien E (APOE) gene, the cholesterol ester transfer protein (CETP) gene and the hepatic triglyceride lipase (LIPC) gene in 383 individuals from 100 Korean families. The genotype was determined by Orchid’s SNP-ITTM technology. The association analysis of the quantitative traits was performed using the quantitative transmission disequilibrium test. Results: A component analysis of the phenotypic variance explained 24.7% of the genetic variance on the total cholesterol, 26.4% of the genetic variance of the high density lipoprotein (HDL)-cholesterol, 11% of the genetic variance of the triglycerides, 35.6% of the genetic variance of the low density lipoprotein (LDL)-cholesterol and 18.9% of the genetic variance of the LDL-C/HDL-C, respectively. The association of the SNPs in the candidate genes explained a major fraction of the genetic phenotypic variance in the LDL-C/HDL-C ratio, but not in the other lipid profiles. The association with SNPs explained 38.5% of the variance for the total cholesterol, 32.2% of the variance for HDLcholesterol and 29.5% of the variance for LDL-cholesterol relative to the polygenic background. An analysis of the contribution of each gene to the genetic variance showed that ABCA1, APOE, CETP and LIPC influenced the variations in total cholesterol, LDL-cholesterol and LDL-C/HDL-C. The variation in HDL-cholesterol was influenced by ABCA1, APOA5 and APOE. Conclusion: We identified that the genetic variance for the total cholesterol, HDL-cholesterol and LDL-cholesterol, and the LDL-C/HDL-C ratio was significantly influenced by the genetic polymorphisms in 5 candidate genes in the Korean population. Further studies are necessary to identify other genes that can explain a major fraction of the genetic variance for the lipid levels.