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유아교사의 직무요인이 직무만족에 미치는 영향에 관한 연구
김경숙(Kyung ?Sook Kim) 한국컴퓨터정보학회 2015 韓國컴퓨터情報學會論文誌 Vol.20 No.3
본 연구는 유아교사의 직무요인이 직무만족에 미치는 연구로 기관유형에 따른 조절효과분석을 통해 시사점을 도출하는데 연구목적을 두었다. 연구목적을 달성하기 위하여 선행연구를 토대로 유아교사의 직무만족에 대한 변인으로 교사효능감, 감성지능, 동료관계, 보상체계의 관계에 대한 인과모형을 설정한 다음 설문조사를 통하여 실증적으로 분석하였다. 본 연구결과는 유아교사의 교사효능감, 감성지능, 동료관계, 보상체계가 직무만족에 미치는 영향에서는 보상체계가 가장 높게 영향을 미치는 것을 알 수 있었고, 감성지능, 동료관계, 교사효능감의 순으로 나타났다. 기관유형에 따른 조절효과 분석에서는 감성지능은 유치원과 어린이집에 따라 유의미한 차이가 있는 것으로 나타났으며, 교사효능감, 동료관계, 보상체계는 유의미한 차이가 없는 것으로 나타났다. 이러한 연구결과를 바탕으로 유아교사의 직무만족을 효과적으로 달성할 수 있는 방안 모색의 기초자료를 제공하는데 있다. This study aims to identify the effects of job factors related to children’s teachers on job satisfaction and present suggestions through an analysis of the modulating effects according to the type of institute. In order to achieve this purpose, this study set up a cause and effect model on teacher’s efficacy, emotional intelligence, friendship, and a reward system as factors of job satisfaction and analysed them based on interviews using a questionnaire. To look into the results of the analysis, it was discovered that a reward system had the greatest effect on job satisfaction for the subjects, followed by emotional intelligence, friendship, and teacher’s efficacy. In an analysis of modulating effects according to the type of institute, the effect of emotional intelligence differed significantly depending on kindergartens and day care centers. Teacher’s efficacy, friendship and a reward system were not significantly different. Based on the results above, this study provides basic data for the development of strategies for children’s teachers to effectively achieve job satisfaction.
Analysis of Vaspressin-Induced Ca 2+ Increase in Rat Hepatocytes
Hyun-SookKim,FumikazuOkajima,DongSoonIm 대한약학회 2003 Archives of Pharmacal Research Vol.26 No.1
To analyze vasopressin-induced Ca2+ increase in liver cells, rat hepatocytes were isolated and attached to collagen-coated cover slips. Using fura-2, a Ca2+-sensing dye, changes in intracellular Ca2+ concentration by vasopressin were monitored. Results in this communication suggested that vasopressin-induced Ca2+ increase were composed of both Ca2+ release from internal Ca2+ stores and influx from the plasma membrane. The Ca2+ influx consisted of two distinguishable components. One was dependent on the presence of vasopressin and the other was not. SK&F96365 blocked vasopressin-induced Ca2+ influx in a dose-dependent manner. Vasopressin-induced Ca2+ release from internal stores diminished in a primary culture of hepatocytes according to the culture time. However, changes in vasopressin-induced Ca2+ influx across the plasma membrane differed from changes in the Ca2+ release from internal stores, suggesting two separate signalings from receptor activation to internal stores and to the plasma membrane.