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Benefits of contrast-enhanced ultrasonography for interventional procedures
Constantin Arndt Marschner,Johannes Rübenthaler,Matthias Frank Froelich,Vincent Schwarze,Dirk-André Clevert 대한초음파의학회 2021 ULTRASONOGRAPHY Vol.40 No.2
For evaluating unclear tumorous lesions, contrast-enhanced ultrasonography (CEUS) is an important imaging modality in addition to contrast-enhanced computed tomography and magnetic resonance imaging, and may provide valuable insights into the microvascularization of tumors in dynamic examinations. In interventional procedures, CEUS can make a valuable contribution in pre-, peri-, and post-interventional settings, reduce radiation exposure and, under certain circumstances, decrease the number of interventions needed for patients.
THE NEW GENERATION OF THE BMW CHILD SEAT AND OCCUPANT DETECTION SYSTEM SBE 2
Lu, Yan,Marschner, Christian,Eisenmann, Lutz,Sauer, Sivart The Korean Society of Automotive Engineers 2002 International journal of automotive technology Vol.3 No.2
A new generation of the BMW child seat and occupant detection system SBE2 far a smart airbag system is described. The SBE2 system consists of two subsystems: OC (Occupant Classification) and FDS (Field Detection System). The OC system is a force sensitive sensor array that measures a pressure profile. The FDS system detects child seat and occupant according to the change of electrical field generated by four capacitive plates. Combining the signals from both subsystems, the BMW SBE2 system can distinguish fully automatically between a child seat and a person.
THE NEW GENERATION OF THE BMW CHILD SEAT AND OCCUPANT DETECTION SYSTEM SBE 2
Yan Lu,Christian Marschner,Lutz Eisenmann,Sivart Sauer 한국자동차공학회 2002 International journal of automotive technology Vol.3 No.2
A new generation of the BMW child seat and occupant detection system SBE2 for a smart airbag system is described. The SBE2 system consists of two subsystems: OC (Occupant Classification) and FDS (Field Detection System). The OC system is a force sensitive sensor array that measures a pressure profile. The FDS system detects child seat and occupant according to the change of electrical field generated by four capacitive plates. Combining the signals from both subsystems, the BMW SBE2 system can distinguish fully automatically between a child seat and a person.
Immune mechanisms in the different phases of acute tubular necrosis
( Fedor Kundert ),( Louise Platen ),( Takamasa Iwakura ),( Zhibo Zhao ),( Julian A. Marschner ),( Hans-joachim Anders ) 대한신장학회 2018 Kidney Research and Clinical Practice Vol.37 No.3
Acute kidney injury is a clinical syndrome that can be caused by numerous diseases including acute tubular necrosis (ATN). ATN evolves in several phases, all of which are accompanied by different immune mechanisms as an integral component of the disease process. In the early injury phase, regulated necrosis, damage-associated molecular patterns, danger sensing, and neutrophil-driven sterile inflammation enhance each other and contribute to the crescendo of necroinflammation and tissue injury. In the late injury phase, renal dysfunction becomes clinically apparent, and M1 macrophage-driven sterile inflammation contributes to ongoing necroinflammation and renal dysfunction. In the recovery phase, M2-macrophages and anti-inflammatory mediators counteract the inflammatory process, and compensatory remnant nephron and cell hypertrophy promote an early functional recovery of renal function, while some tubules are still badly injured and necrotic material is removed by phagocytes. The resolution of inflammation is required to promote the intrinsic regenerative capacity of tubules to replace at least some of the necrotic cells. Several immune mechanisms support this wound-healing-like re-epithelialization process. Similar to wound healing, this response is associated with mesenchymal healing, with a profound immune cell contribution in terms of collagen production and secretion of pro-fibrotic mediators. These and numerous other factors determine whether, in the chronic phase, persistent loss of nephrons and hyperfunction of remnant nephrons will result in stable renal function or progress to decline of renal function such as progressive chronic kidney disease.