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JuHwan Lim,GyoungBum Kim,SungWoo Hwang IEEE 2008 IEEE microwave and wireless components letters Vol.18 No.1
<P>Suppression of microwave resonances in wirebond transitions is reported. The transition is made between conductor-backed coplanar waveguides on two different substrates (alumina and silicon), and multiple wirebonds connecting two grounds on each substrate are shown to remove the resonances. It is shown that this additional wirebonding eliminates the nodes which are responsible for the unwanted resonant modes with the frequencies far below the first resonant frequency (19 GHz) of the single substrates. Our work is a unique demonstration of using multiple wirebonds for the removal of resonances.</P>
Opposite Localization of Luteinizing Hormone Receptors and Galectin-3 in Mature Mouse Ovaries
Juhwan Kim(김주환),Miyoung Yang(양미영),Sung-Ho Kim(김성호),Jong-Choon Kim(김종춘),Seungjoon Kim(김승준),Hong-Gu Joo(주홍구),Taekyun Shin(신태균),Changjong Moon(문창종) 한국생명과학회 2012 생명과학회지 Vol.22 No.5
본 연구는 성숙마우스의 난소에서 황체형성호르몬수용체(luteinizing hormone receptor, LHR)와 beta-galctoside-binding animal lectin인 galetin-3 (Gal-3)의 발현분포를 면역조직화학방법으로 평가하였다. LHR은 활동(성숙) 황체에서 강하게 발현되는 반면에, Gal-3의 발현은 퇴행중인 황체와 폐쇄난포에서 높게 나타났다. 또한, 임신중인 마우스의 황체에서는 LHR의 면역반응이 강하게 나타났으나, Gal-3의 면역반응은 현저히 낮았다. 위의 결과를 종합해 볼 때, 성숙마우스의 난소에서 LHR과 Gal-3는 난소의 특정한 시기에 따라 다른 분포양상을 나타냄을 알 수 있었으며, 이들 각각의 단백질의 발현으로 보아 황체의 형성과 퇴행에서 상반된 역할을 할 것으로 여겨진다. Received January 5, 2012 /Revised April 12, 2012 /Accepted May 8, 2012 The present study evaluated the localization of luteinizing hormone receptors (LHR) and galectin-3 (Gal-3), a beta-galactoside-binding animal lectin, in the mature mouse ovaries by immunohistochemical analysis. Intense LHR immunoreactivity was detected in the active corpus luteum (CL), whereas expression of Gal-3 was high in the regressing CL and atretic follicle. In the CL of pregnant mice, LHR immunoreactivity was intense, but Gal-3 expression was low. Thus, LHR and Gal-3 had opposite patterns of expression in mature mouse ovaries, suggesting that both proteins have stage-specific expression patterns and are possibly involved in CL formation and regression.
( Juhwan Lee ),( Sieun Park ) 대한물리치료학회 2015 대한물리치료학회지 Vol.27 No.4
Purpose: This study was conducted to investigate the effect of cigarette smoking on physical fitness (dominant hand grip power, dominant isokinetic leg muscle strength, abdominal muscle endurance, flexibility, cardiopulmonary endurance) and depression in patients with chronic low back pain. Methods: This study was a cross sectional study and subjects consisted of 60 young males with chronic low back pain. The subjects were allocated to two groups following a self-report survey: cigarette smoking group (n=25) or non-smoking group (n=35). Physical fitness (dominant hand grip power, dominant isokinetic leg muscle strength, abdominal muscle endurance, flexibility, cardiopulmonary endurance) were measured using objective methods and depression in patients with chronic low back pain was measured using the Korean version of center for epidemiologic studies depression (CES-D) scale. Results: The results of this study were as follows: In physical fitness, the cigarette smoking group showed a significant decrease in abdominal muscle endurance, flexibility, and cardiopulmonary endurance compared with the non-smoking group. Depression index (CES-D scale score) was significantly higher in the cigarette smoking group than in the non-smoking group. Conclusion: These results suggest that cigarette smoking had a negative effect on abdominal muscle endurance, flexibility, and cardiopulmonary endurance in patients with chronic low back pain. In addition, depression of patients with chronic low back pain was affected by cigarette smoking. Thus, we suggested that cigarette smoking may play a significant role in the deterioration of physical fitness and depression of chronic low back pain patients.
Residential radon and environmental burden of disease among Non-smokers
Juhwan Noh,Jungwoo Sohn,Jaelim Cho,Dae Ryong Kang,Sowon Joo,Changsoo Kim,Dong Chun Shin 대한직업환경의학회 2016 대한직업환경의학회지 Vol.28 No.-
Background: Lung cancer was the second highest absolute cancer incidence globally and the first cause of cancer mortality in 2014. Indoor radon is the second leading risk factor of lung cancer after cigarette smoking among ever smokers and the first among non-smokers. Environmental burden of disease (EBD) attributable to residential radon among non-smokers is critical for identifying threats to population health and planning health policy. Methods: To identify and retrieve literatures describing environmental burden of lung cancer attributable to residential radon, we searched databases including Ovid-MEDLINE, -EMBASE from 1980 to 2016. Search terms included patient keywords using ‘lung’ , ‘neoplasm’ , exposure keywords using ‘residential’ , ‘radon’ , and outcomes keywords using ‘years of life lost’ , ‘years of life lost due to disability’ , ‘burden’. Searching through literatures identified 261 documents; further 9 documents were identified using manual searching. Two researchers independently assessed 271 abstracts eligible for inclusion at the abstract level. Full text reviews were conducted for selected publications after the first assessment. Ten studies were included in the final evaluation. Review: Global disability‐adjusted life years (DALYs)(95 % uncertainty interval) for lung cancer were increased by 35.9 % from 23,850,000(18,835,000-29,845,000) in 1900 to 32,405,000(24,400,000-38,334,000) in 2000. DALYs attributable to residential radon were 2,114,000(273,000-4,660,000) DALYs in 2010. Lung cancer caused 34,732,900(33,042,600 ~ 36,328,100) DALYs in 2013. DALYs attributable to residential radon were 1,979,000(1,331,000-2,768,000) DALYs for in 2013. The number of attributable lung cancer cases was 70-900 and EBD for radon was 1,000-14,000 DALYs in Netherland. The years of life lost were 0.066 years among never-smokers and 0.198 years among ever-smoker population in Canada. Conclusion: In summary, estimated global EBD attributable to residential radon was 1,979,000 DALYs for both sexes in 2013. In Netherlands, EBD for radon was 1,000–14,000 DALYs. Smoking population lost three times more years than never-smokers in Canada. There was no study estimating EBD of residential radon among never smokers in Korea and Asian country. In addition, there were a few studies reflecting the age of building, though residential radon exposure level depends on the age of building. Further EBD study reflecting Korean disability weight and the age of building is required to estimate EBD precisely.
The environmental burden of lung cancer attributable to indoor radon exposure in Korea
Juhwan Noh(노주환),Jungwoo Sohn(손정우),Heeseon Jang(장희선),Seong-Kyung Cho(조성경),Yun Tae Kim(김윤태),Eung Joo Park(박응주),Dae Ryong Kang(강대용),Dong Chun Shin(신동천),Changsoo Kim(김창수) 환경독성보건학회 2018 한국독성학회 심포지움 및 학술발표회 Vol.2018 No.6