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<천금방(千金方)>의 침구처방용혈(鍼灸處方用穴)의 일반규률(一般規律) 연구(硏究)
김연진 ( Yun Jin Kim ),김재효 ( Jae Hyo Kim ),이충인 ( Zhong Ren Li ),손인철 ( In Chel Sohn ) 대한경락경혈학회 2005 Korean Journal of Acupuncture Vol.22 No.4
Objective: <千金方(Qianjinfang)> which documents a great amount of prescription of Acupuncture and moxibustion takes an important role during the development of acupuncture and moxibustion. Thus, it have given rise us to the research-situation of Tang Dynasty China of previous period. This study was to outline the rules of choosing acupoints as well as the prescriptions of contemporary times in reference with <千金方(Qianjinfang)>, and to discuss the conception of acupuncture and moxibustion in <千金方(Qianjinfang)>. Method: Based on <千金方(Qianjinfang)>, the parameters were categorized to a certain scope, syndromes in different type were classified. Also numbers of acupoints, route of meridians locations, utilities of special acupoints even prescription methods were statistically analyzed in reference with <千金方(Qianjinfang)>. Results & Conclusion: Acupuncture prescription in <千金方(Qianjinfang)> was mostly presented by single acupoint and this was basic prescription of its rule of choosing acupoints. Choosing acupoint for the majority of various diseases started from choosing meridians with the disorder, but no rules for choosing acupoints was clear. There was basically various methods in choosing acupoints, but little were used by Biao-li meridian choosing method (表裏經配穴). In the high frequency of use, wu-shu acupoints (五수穴) was higher used than other specific acupoints (特定穴). While wu-shu acupoints (五수穴) treated the major parts of the diseases, shu-mo acupoints (수募穴) were strictly used on system. Consequently, The theoretical basis originating the choosing rules of the acupoints and prescriptions was shown in <千金方(Qianjinfang)> as representative clinical reference, through which it was valuable to analyze the prescription rule and specificity of the acupoints.
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대방풍탕(大防風湯) 약침(藥鍼)이 Carrageenan으로 유도한 관절염에 미치는 영향(影響)
구성태 ( Sung Tae Koo ),황재호 ( Jae Ho Hwang ),송문영,박유리,손인철 ( In Chel Sohn ),김경식 ( Kyoung Sik Kim ) 대한경락경혈학회 2005 Korean Journal of Acupuncture Vol.22 No.3
Objectives: DaeBangPungTang(DBPT) is one of the prescriptions used for the treatment of knee arthritis in oriental medicine. The present study aimed to examine the analgesic effect of DBPT on a rat model of carrageenan-induced arthritis, and the relations between DBPT-induced analgesia and endogenous nitric oxide(NO) and inducible NO synthase (iNOS), cyclooxygenase-2 (COX-2), and c-Fos protein expression in the spinal cord. Methods: Carrageenan-induced arthritis rat model was used to test the effect of DBPT as a chronic pain model. After the induction of arthritis, rats subsequently showed a reduced stepping force of the affected limb for at least the next 4 days. The reduced stepping force of the limb was presumably due to a painful knee, since oral injection of indomethacin produced temporary improvement of weight bearing. DBPT dissolved in normal saline was injected several acupoints. After the treatment, behavioral tests measuring stepping force were periodically conducted during the next 12 hours. Results: DBPT produced significant improvement of stepping force of the hindlimb affected by the arthritis lasting at least 9 hours. The magnitude of this improvement was equivalent to that obtained after an oral injection of 3 mg/kg of indomethacin and this improvement of stepping force was interpreted as an analgesic effect. DBPT produced the improvement of stepping force of the affected hindlimb in a dose-dependent manner. Both NO production and iNOS, COX-2 protein expression increased by arthritis were suppressed by DBPT. DBPT on combination with electroacupuncture (EA) produced more powerful and longer lasting improvement of stepping force of the hindlimb affected by the arthritis than either DBPT or EA did. Conclusion: The present study suggest that DBPT produces a potent analgesic effect on the chronic knee arthritis pain model in the rat and that DBPT-induced analgesia modulate endogenous NO through the suppression of iNOS/COX-2 protein expression.
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