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- 저자 : ( Seirhan Kim ) (검색결과 4 건)
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Kim, Young-Gun,Kim, Seirhan,Han, Seung Jin,Kim, Dae Jung,Lee, Kwan-Woo,Kim, Hae Jin Hindawi 2018 Journal of diabetes research Vol.2018 No.-
<P><B>Background</B></P><P> Information on the risk of acute pancreatitis in patients receiving dipeptidyl-peptidase IV inhibitors (DPP-4i) is limited and controversial. One study suggested that the differences in findings between these meta-analyses were attributed to whether they included large randomized control trials with cardiovascular outcomes or not. The aim of our study was to determine whether the use of DPP-4i increases the risk of acute pancreatitis compared with sulfonylurea (SU) and whether the risk is higher in patients with underlying cardiovascular disease (CVD). </P><P><B>Methods</B></P><P> A population-based cohort study was performed using Korean National Health Insurance Service-National Sample Cohort data. We included 33,395 new users of SU and DPP-4i from 1 January 2008 to 31 December 2015. SU-treated patients and DPP-4i-treated patients were matched by 1 : 1 propensity score matching. We used Kaplan–Meier curves and Cox proportional hazards regression analysis to calculate the risk of acute pancreatitis. </P><P><B>Results</B></P><P> The hazard ratio (HR) of hospitalization for acute pancreatitis was 0.642 (95% confidence interval (CI): 0.535–0.771) in DPP-4i-treated patients compared with SU-treated patients. The HR of DPP-4i use was also lower than that of SU use in patients without underlying CVD (HR: 0.591; 95% CI: 0.476–0.735) but not in patients with underlying CVD (HR: 0.727; 95% CI: 0.527–1.003). </P><P><B>Conclusion</B></P><P> Our findings suggest that DPP-4i is less likely to cause drug-induced pancreatitis than SU. This finding was not evident in patients with CVD, but DPP-4i was not more likely to induce pancreatitis in these patients than SU was.</P>
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Comparison of estimated glomerular filtration rate equations at the time of hemodialysis initiation
( Min Jeong Lee ),( Seirhan Kim ),( Inwhee Park ),( Heung Soo Kim ),( Gyu Tae Shin ) 대한신장학회 2015 Kidney Research and Clinical Practice Vol.34 No.4
Background: Estimated glomerular filtration rate (eGFR) is one of the most important guidelines in deciding the optimal timing of dialysis initiation. In the present study, we calculated the eGFR at the time of hemodialysis (HD) initiation using 5 commonly used equations to relate them with clinical and laboratory characteristics of the patients and to evaluate which of these equations best define the eGFR at HD initiation. Methods: We retrospectively analyzed 409 end-stage renal disease patients who were newly started on HD treatment in our institution. The eGFR was calculated using the CockcrofteGault equation, the CockcrofteGault equation corrected for body surface area, the Modification of Diet in Renal Disease (MDRD) equation, the Chronic Kidney Disease Epidemiology Collaboration (CKD-EPI) equation, and the Nankivell equation. Results: The mean eGFRs at HD start were significantly different across the equations. The mean eGFR was 7.8 mL/min for the corrected CockcrofteGault equation, 7.7 mL/min for the CockcrofteGault equation, 6.2 mL/min/1.73 m2 for the MDRD equation, and 5.6 mL/min/1.73 m2 for the CKD-EPI equation. The corrected Cockcroft eGault, the MDRD, and the CKD-EPI equations were well correlated with all CKDspecific complications including hypertension, anemia, hyperkalemia, metabolic acidosis, hypocalcemia, hyperphosphatemia, and hyperparathyroidism. The mean eGFR calculated by the corrected CockcrofteGault equation showed the lowest coefficient of variation among all the equations. Conclusions: The eGFR at HD initiation are significantly different according to the used eGFR equations, and the corrected CockcrofteGault equation may be the best in defining the eGFR at HD initiation.
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( Eunsoo Lim ),( Sunah Hyun ),( Jae Myeong Lee ),( Seirhan Kim ),( Min-jeong Lee ),( Sun-mi Lee ),( Ye-sung Oh ),( Inwhee Park ),( Gyu-tae Shin ),( Heungsoo Kim ),( Donald E. Morisky ),( Jong Cheol Je 대한신장학회 2018 Kidney Research and Clinical Practice Vol.37 No.1
Background: For phosphate control, patient education is essential due to the limited clearance of phosphate by dialysis. However, well-designed randomized controlled trials about dietary and phosphate binder education have been scarce. Methods: We enrolled maintenance hemodialysis patients and randomized them into an education group (n = 48) or a control group (n = 22). We assessed the patients’ drug compliance and their knowledge about the phosphate binder using a questionnaire. Results: The primary goal was to increase the number of patients who reached a calcium-phosphorus product of lower than 55. In the education group, 36 (75.0%) patients achieved the primary goal, as compared with 16 (72.7%) in the control group (P = 0.430). The education increased the proportion of patients who properly took the phosphate binder (22.9% vs. 3.5%, P = 0.087), but not to statistical significance. Education did not affect the amount of dietary phosphate intake per body weight (education vs. control: -1.18 ± 3.54 vs. -0.88 ± 2.04 mg/kg, P = 0.851). However, the dietary phosphate-to-protein ratio tended to be lower in the education group (-0.64 ± 2.04 vs. 0.65 ± 3.55, P = 0.193). The education on phosphate restriction affected neither the Patient-Generated Subjective Global Assessment score (0.17 ± 4.58 vs. -0.86 ± 3.86, P = 0.363) nor the level of dietary protein intake (-0.03 ± 0.33 vs. -0.09 ± 0.18, P = 0.569). Conclusion: Education did not affect the calcium-phosphate product. Education on the proper timing of phosphate binder intake and the dietary phosphate-to-protein ratio showed marginal efficacy.
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Severe but reversible acute kidney injury resulting from Amanita punctata poisoning
( Eun Jung Kang ),( Ka Young Cheong ),( Min Jeong Lee ),( Seirhan Kim ),( Gyu Tae Shin ),( Heung Soo Kim ),( In Whee Park ) 대한신장학회 2015 Kidney Research and Clinical Practice Vol.34 No.4
Mushroom-related poisoning can cause acute kidney injury. Here we report a case of acute kidney injury after ingestion of Amanita punctata, which is considered an edible mushroom. Gastrointestinal symptoms occurred within 24 hours from the mushroom intake and were followed by an asymptomatic period, acute kidney injury, and elevation of liver and pancreatic enzymes. Kidney function recovered with supportive care. Nephrotoxic mushroom poisoning should be considered as a cause of acute kidney injury.
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