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RISS 인기검색어
- 검색키워드
- 저자 : 한심석 ( Shim Suck Hahn ) (검색결과 7 건)
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한심석 ( Shim Suck Hahn ),민영일 ( Y. I. Min ),고광욱 ( K. W. Koh ) 대한내과학회 1970 대한내과학회지 Vol.13 No.5
The acid-base status was evaluated in thirty-four cases of various liver diseases with hepatic encephalopathy using the Astrup method. The results were analysed in regard to the disease group, conscious state and prognosis as well as comparison with the la
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Stilbazium Iodide 를 이용한 장내 기생충증 특히 편충의 구충 효과에 관한 실험적 연구
한심석 ( Shim Suck Hahn ),박실무 ( Sill Moo Park ) 대한내과학회 1970 대한내과학회지 Vol.13 No.2
Clinical trial on the anthelmintic effect of the Stilbazium Iodide, a new pyridine derivative, was performed in 20 selected cases with intestinal helminthiasis at the Department of Internal Medicine in Seoul National University Hospital. Stilbazium Iodide
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吳仁赫,韓沁錫 서울대학교 1957 서울대학교 論文集 Vol.6 No.-
To examine the adrenocortical function of the patients with liver diseases, the author applied a modification of Thorn's test, intravenous injection of 2.5 mgm of ACTH, to 12 cases of clonorchiasis, 5 of hepatits, 5 of liver cirrhosis and 5 normal individuals as a control. The result of the examination were as following: 1) The eosinopenic response following the administration of ACTH was not distinct in the cases with hepatic dysfunction. 2) On the other hand, in the cases with normal hepatic function, the eosinopenic response was within normal limits.
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간실질세포의 손상이 철흡수에 미치는 영향에 관한 실험적 연구
김목현,한심석 대한핵의학회 1971 핵의학 분자영상 Vol.5 No.2
Since the iron balance is maintained by regulated intestinal absorption rather than regulated excretion, there have been many reports concerning the factors which may influence the intestinal iron absorption. As the liver is the largest iron storage organ of the body, any hepatocellular damage may result in disturbances in iron metabolism, e,g., frequent co-existence of hemochromatosis and liver cirrhosis, or elevated serum iron level and increased iron absorption rate in patients with infectious hepatitis or cirrhosis. In one effort to demonstrate the influence of hepatocellular damage on intestinal iron absortion, the iron absorption rate was measured in the rabbits whose livers were injured by a single subcutaneous injection of carbon tetrachloride (doses ranging from 0.15 to 0.5cc per kg of body weight) or by a single irradiation of 2, 000 to 16, 000 rads with Co on the liver locally. A single oral dose of 1μCi of Fe-citrate with 0. 5mg of ferrous citrate was fed in the fasting state, 24 hours after hepaic damage had been induced, without any reducing or chelating agents, and stool was collected for one week thereafter. Serum iron levels, together with conventional liver function teats, were measured at 24, 48, 72, 120 and 168 hours after liver damage had been induced. All animals were sacrificed upon the completing of the one week's test period and tissue specimens were prepared for H-E and Gomori's iron stain. Following are the results. 1. Normal iron absorption rate of the rabbit was 41.72±3.61% when 0.5mg of iron was given in the fasting state, as measured by subtracting the amount recovered in stool collected for 7 days from the amount given. The test period of 7 days is adequate, for only 1% of the iron given was excreted thereafter. 2. The intestinal iron absorption rate and serum iron level were significantly increased when the animal was poisoned by a single subcutaneous injection of 0.15cc, per kg. of body weight of carbon tetrachloride or more, or the liver was irradiated with a single dose of 12000 rads or more. The results of liver function tests which done simultaneously remained within normal limit except SGOT and SGPR which were somewhat increased. 3. In each case, there has been good correlation between the extent of liver cell damage and degree of increased iron absorption rate or serum iron level. 4. The method of liver damage appeared to make no obvious difference in the pattern of iron deposit in liver. This may be partly due to the fact that tissue specimens were obtained too late, for by this time the elevated serum iron level had returned within normal range and the pathological changes were almost healed. 5. The possible factors and relationship between intestinal iron absorption and hepatic parenchymal cell damage has been discuseed.
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FUNKTIONSPRUEFUNG DER LEUKOPOESE DES KNOCHENMARKS(I. MITTEILUNG)
Lee, Munho,Hahn, Chin Kwan,Hahn, Shim Suck 서울대학교 1959 서울대학교 論文集 Vol.8 No.-
Pyrifer를 使用하여 人工的으로 發熱을 일으킴으로써 白血球의 量的變化를 觀察한 Hoff 及共同硏究者의 動物實驗을 基礎로하여 Moeschlin(1954)은 Pyrifer을 骨髓機能檢査法에 使利用하여 좋
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Oh, In Hyug,Lee, Munho,Hahn, Shim Suck 서울대학교 1959 서울대학교 論文集 Vol.8 No.-
血液凝固過程의 第二相에서 가장 重要한 部分을 차지하고있는 Prothrombin이 主로 肝臟에서 生成된다는 點과 이 Prothromibin이 Vitamin K 存在下에서 生成된다는 點等으로 미루어 肝臟疾患과 Prothrombin値와의 關係 및 Vitamin K 投與에 依한 Prothrombin 値의 消長을 測定한 目的으로 著者들은 서울大學病院內科 外來 及 入院患者中에서 肝硬變症을 爲始하여 數種의 肝臟病患者를 選擇하여 Quick氏의 Original (onestage) method에 依하여 ProthrombinKomplex値를 測定하고 또 肝硬變症과 閉塞性黃疸患者中 ProthrombinKomplex値가 正常値以下인 症例에 對하여는 Koller氏의 VitaminK 投與法을 適用하여 그 反應을 檢査하였다。卽충肝硬變症 30例, 閉塞性黃疸 14例, 肝癌 14例, 肝吸 症 9例, 肝炎 3例, 及 肝內膽管炎 3例에 對하여 各各 Quick test를 施行한 結果 1) 肝硬變症群에 있어서는 Quick値는 肝機能障碍度가 甚할수록 低下率도 顯著한 傾向을 보았으며. 2) 閉塞性黃疸群에 있어서는 Quick値가 一般的으로 低下되어있기는 하나 Quick値는 그 活疸의 黃度와는 比例되지 않으며 長期間에 걸친 閉塞性黃疸로 因하여 實質障碍가 發生하였다고 생각되는 症例에서는 Qwick 値의 低下가 顯著하였고, 3) 其他의 肝疾患 卽 肝癌, 肝炎, 肝內膽管炎 肝吸충症에있어서는 Quick 値는 肝機能障碍가 있는 症例에서 低下되어있음을 보았다。 4) Koller氏法에 依한 Vitamin K 投與에 對한 Quick値의 反應을 보기 爲하여 肝硬變症 及 閉塞性黃疸患者中에서 Quick値가 正常値以下인 症例를 選擇하여 肝硬變症 20例, 閉塞性黃疸 10例에서 實驗해본 結果, 肝硬變症群에 있어서는 그 反應이 全無 惑은 輕微하였으나 閉塞性黃疸群에 있어서는 그反應이 全例를 通하여 艮好함을 보았다。 Im Vorliegenden werden die Resultate, die mit Quick’scher Methods und von Keller angegebenen VitaminKTest bei 73 Patienten mit verschiedenen Lebererkrankungen erhalten wurden, mitgeteilt. Mit dieser Methoden ergeben sich folgende Schlussfolgerungen: Fuer die Lebererkrankungen spricht: Fast in allen Faellen sinken die Quick’schen Werte ab, die mit den Leberschaedigungen parallel laufen. Bei schweren Lebercirrhosen finden sich eine VitaminK refraktaere Verminderung von Prothrombinkomplexe und geringe Ansprechen auf Vitamin K. Der von Koller eingefuehrte Leberfunktionstest mit Vitamin K erlaubt die Unterscheidung zwischen Verschlussikterue und Parenchymikterus.
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