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HBsAg 양성 급성 간염에서 간조직내 B 형 간염 바이러스 항원 검색의 임상적 의의
서상렬(Sang Yeol Suh),서동진(Dong Jin Suh) 대한내과학회 1989 대한내과학회지 Vol.37 No.2
N/A To evaluate the clinical significance of hepatitis B virus antigens in liver in HBsAg positive acute viral hepatitis, HBsAg end HBcAg were detected by the ABC immunoperoxidase method in 39 paraffin embedded liver tissues with histologic findings of acute viral hepatitis, and their correlation with serologic markers of HBV was studied. The results were as follows; 1) Among 39 patients with HBsAg positive acute viral hepatitis, hepatitis B virus antigens in liver tissue were demonstrated in 22 patients (56.4%). Of the 22 patients, both HBsAg and HBcAg were demonstrated in 7 patients, HBsAg only in 11 patients and HBcAg in 4 patients. 2) IgM anti-HBc was positive in 15 (38.4%)of the 39 patients. While IgM anti-HBc was positive in 2 (9%) among 22 tissue antigen positive patients, 13 (76.5%)of the 17 patients without tissue antigen had IgM anti- HBc. 3) Serum HBV-DNA was positive in 69.2%. While HBV-DNA was positive in 86.7% of tissue antigen positive patients, 54.5% of patients without tissue antigen did not have HBV-DNA. The level of serum HBV-DNA was significantly higher in tissue antigen positive patients (30.1±38.9pg/ml) than in negative patients (5.2±3.4pg/ml). 4) Twenty-seven patients were followed over 6 months. Although HBsAg was seroconverted in S (88.9%) among 9 patients without tissue antigen, all patients who had hepatitis B virus antigens in the liver had persistent hepatitis B surface antigenemia over 6 months. The presence of HBV antigens in the liver in acute HBsAg positive hepatitis appears to correspond with acute exacerbation of chronic HBV infection or superinfection with non-B hepatitis virus in HBU carriers rather than acute HBV infection, since most of them have no IgM anti-HBc and have chronic antigenemia. Such conditions may occur frequently in Korea where the HBsAg carrier rate is high.
김윤석,서상렬,한병철,김방수,조진수 대한내과학회 1996 대한내과학회지 Vol.50 No.4
Two cases of vitamin B12 deficiency megaloblastic anemia are presented and literature for the cases are briefly reviewed. The first case is a 54-year-old man who was diagnosed as gastric ulcer perforation, and subtotal gastrectomy with gastrojejunostamy were performed 10 years ago. The second case is a 78-year-old man complicated with intrinsic factor deficiendcy and atrophic gastritis. Both cases were admitted to our hospital because of anemic symptoms such as dizziness, headache, tinnitus, general weakness, and appetite loss. Peripheral blood revealed thrombocytopenia, macrocytic anemia with anisocytosis and hypersegmented neutrophil, and bone marrow findings were decreased M:E ratio and increased cellularity. Schilling test revealed the excretion of 0.51% and 1.7% in 24hr urine collection (control 7%) respectly, and serum vitamin B12 level was decreased markedly. After treatment of parenteral vitamin B12, their clinical symptoms and hematologic findings improved progressively and they have been followed up on outpatient basis with maintenance therapy of vitamin B12.
본태성고혈압 및 만성신부전에서 요 Prostaglandin E₂의 의의(義意)
김형규,변관수,서상렬,남궁호삼,흥순국 대한신장학회 1984 Kidney Research and Clinical Practice Vol.3 No.2
In order to define tke changes of urine prosta- glandin E in chronic renal failure, urine excre-tion of prostaglandin E, was measured ' in 24 normal control group, in 23 essential hypertension not associated with renal impairment, and in 25 chronic renal failure associated witn hypertension of which creatinine clearance was below 20 ml/ min. Otherwise serum and urine sodium, serum creatinine and creatinine clearance were measured. The results obtained are as follows; 1) The value of urine prostaglandin E, in nor- mal control group is 367.1±168.24(mean S.D.) pg/ml., in essential hypertension 350.4±97.32 pg/ ml. And in chronic renal failure with hyperte nsion 84. 5±40. 76 pg/ml. Respectively. In chronic renal failure group, urine prostaglandin E, is significantly lower than in normal control group (P$lt;0. 01). 2) Urine prostaglandin E, has no significant relation to diastolic blood pressure, serum and urine sodium and creatinine clearance in each group. 3) Urine prostaglandin E, is not reIated to sex ange distribution. In our results, it is cansidered that the decrease of urine prostaglandin E in chronic renal failure is accompanied by the progressrve reduction of renal functioning mass.
DSP에 기초한 모터 속도 제어용 2자유도 제어기 구현을 위한 시뮬레이션
최우석(Wooseok Choi),서상렬(Sang Yeol Seo),이세한(Se-Han Lee) 대한기계학회 2006 대한기계학회 춘추학술대회 Vol.2006 No.6
In this research a simulation for implementing 2-D.O.F. motor speed controller based on DSP is carried out. By using the reference input and plant output, a 2-D.O.F. controller is expected to show better control performance than a simple feedback controller that is using only the difference information between the reference input and plant output. Additionally, it is well known that a 2-D.O.F. controller allows the control system to have better control performance without contaminating its stability. When a DSP is used for motor control, the 2-D.O.F. control scheme is easily to added to the original controller with adding some lines of program codes. The simulation of the motor speed control is carried out by taking account of input/output accuracy quantization characteristics of a DSP. The resultant program codes may be able to be used for an actual DSP controller.
만성 신부전 환자에서 복막 투석중 발생한 Chyloperitoneum 1예
김영준,이순애,정상용,서상렬,임규철,정종익,김진휘,권덕호 대한신장학회 1998 Kidney Research and Clinical Practice Vol.17 No.4
Chylous ascites is extravasation of lymphatic fluid and retention in the peritoneal cavity due to traurna or obstruction of the lymphatic system. Chylous ascites is very rare complication of Continuous Ambulatory Peritoneal Dialysis(CAPD) and is associated with trauma to the lymphatics during catheter insertion in the early stage of CAPD and repeated mild trauma to the lyrnphatics during long-term dialysis. Chylous ascites in the CAPD is suspected when the drained peritoneal fluid is milky white and confirmed by demonstration of the specific components of chyle, such as elevated triglyceride and low cholesterol than plasma and should be differentiated from pseudochyle and bacterial peritonitis. We report a case of chylous ascites in a patient undergoing CAPD at 2 months later of initiation of CAPD, which was not improved by conservative management. So CAPD catheter was removed and renal replacement therapy was changed to hemodialysis.
비당뇨성 말기신부전환자에서 복막투석 중 발생한 고혈당증 및 고삼투압증후군 1 예
김윤석,이양규,김중근,추장식,서상렬 대한내과학회 1995 대한내과학회지 Vol.48 No.1
In non-diabetic end-stage renal disease patients, blood glucose concentrations have not significantly changed over time on CAPD. But hypergycemia and hyperosmolality may develop with the use of hypertonic dextrose solutions for peritoneal dialysis. Hyperglycemia without ketosis can cause coma, convulsions, shock, or death. Severe disturbances in brain function are a consequence of the high collection of glucose in the extracellular fluids resulting in intracellular dehydration. A 54-year-old male, non-diabetic end-stage renal disease patient, who had been taken CAPD for 16 months, was hospitalized because of drowsy mental state. He used 4.25% glucose solution for peritoneal dialysis 3 times in a day because of edema. He revealed hyperglycemia and hyperosmolality. Serum glucose level and osmolality returned to normal after injection of normal saline and insulin.