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김영홍 대한금속재료학회(대한금속학회) 1980 대한금속·재료학회지 Vol.18 No.1
Slag에 의한 탄소포화철의 탈황속도 기구를 1460℃, CO gas의 환원성 분위기 하에서 조사하였다. Slag로는 SiO₂-CaO-MgO계의 slag가 사용되었으며 탈항속도는 slag의 조성, melt geometry 및 CO gas압력, Si 및 MnO의 첨가량 등을 변화시켜가며 측정하였다. 실험결과에 따르면 탈황반응의 속도는 metal 또는 slag중의 유황의 확산속도에 좌우되는 것이 아니고 metal/slag계면에서의 전기 화학적 반응속도의 지배를 받는 것으로 생각된다. 탈황반응은 세단계를 거쳐 진행된다. 즉 Fe의 Anodic reaction과 CO발생속도에 좌우되는 초기의 가장 신속한 반응단계와 CO발생속도에만 지배를 받는 중간속도의 반응단계 및 SiO₂의 환원반응에 지배를 받는 최후의 가장 완만한 단계를 거쳐 진행된다. The kinetics of desul phurization of carbon saturated iron by CaO-MgO-SiO₂ slag was studied at 1460℃ under CO gas atmosphere varying the slag composition, melt geometry, Si and MnO additions, and ambient pressure. It was shown that the desulphurization reaction is not controlled by either diffusion of sulphur in the metal or the slag but by an electrochemical reaction. Desulphurization takes place in three different reaction stages; the intial fastest stage is dependent anodic reaction of iron transfer and CO evolution, the second medium rate stage is controlled by CO evolution, and the final slowest stage controlled by silica reduction from the silicate slag.
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사료내 불포화지방산 및 아연부족이 흰쥐의 혈청과 주요 조직내 몇가지 화학성분에 미치는 영향
金永洪 慶北大學校 1984 論文集 Vol.38 No.-
A study was conducted as a preliminary experiment for seeking out the way of prevention and treatment of porcine parakeratosis. The relationship between low fat diet, low fat-low zinc diet and porcine parakeratosis was studied by analysing the contents of free unsaturated fatty acid, total free fatty acid, phospholipid, unsaturated fatty acid in phospholipid and zinc in serum, liver, kidney, spleen and pancreas of rats fed with the diet adjusted content of unsaturated fatty acid and zinc (low fat diet and low fat-low zinc diet). Fifteen growing rats were divided into 3 groups. First group were fed low fat-low zinc diet containing 42 ppm of zinc and 3,260 ㎎/100g of unsaturated fatty acids, second group low fat diet containing 100 ppm of zinc and 3,260 ㎎/100g of unsaturated fatty acids, and third group control diet containing 100 ppm of zinc and 4,860 ㎎/l00g of unsaturated fatty acids for 100 days. The results were summarized as follows: 1) Free unsaturated fatty acid, total free fatty acid, unsaturated fatty acid in phospholipid and phospholipid concentrations of the blood serum, liver, kidney, spleen and pancreas of low fat-low zinc group were lower than those of control group. Free unsaturated fatty acid and phospholipid concentrations in blood serum (each of 5.62 ㎎/dl, 0.75 ㎎/mI) were significantly decreased as compared with those of control group (each of 6.47 ㎎/dl, 1. 15 ㎎/ml). Especially free unsaturated fatty acid, total free fatty acid and phospholipid concentrations in pancreas were significantly decreased. 2) Zinc concentrations of the blood serum, liver, kidney, spleen and pancreas of low fat-low zinc group were significantly lower than those of control and low fat groups. 3) Free unsaturated fatty acid, total free fatty acid, unsaturated fatty acid in phospholipid and phospholipid concentrations of low fat group were. lower than those of control ;group, but the free unsaturated fatty acid and phospholipid concentrations of blood serum-and pancreas were significantly decreased as compared with those of control group. 4) Zinc concentrations of low fat group were significantly higher than those of low fat-low zinc group, but there was no significant difference in low. zinc and control groups.
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Prostaglandin $F_2{\alpha}$투여가 임신 Rat의 생식에 미치는 영향
김영홍 한국임상수의학회 1999 한국임상수의학회지 Vol.16 No.2
Pregnant rats were treated at various stages of gestation with prostaglandin analogue, cloprostenol alone or concomitant with HCG to study effects on termination of gestation and plasma estrogen and progesterone. Cloprostenol (90 or 180 mg/kg) was administered alone on 1~3, 4~6, 7~9, 9~11 or 11~13 consecutive days of gestation twice a day and in combination with HCG (50 or 100 IU/day) on days of 1~3 or 7~9 once a day. Rats were autopsied on day 21 of gestation or at 6, 12 or 24 hours after treatment on day 6 or 9, respectively. Cloprostenol was found to be nearly 100% effective in preventing implantation, destroying viable fetuses and causing preimplantation losses, but in early gestation, on days 1-3, there was little effect. And when cloprostenol administered concomitant with HCG, corpora lutea were significantly increased, implantation sites and viable fetuses significantly decreased, and pre-and post-implantation losses significantly increased in most cases. Plasma concentrations of estradiol and progesterone were significantly decreased by administering cloprostenol, and estradiol concentration significantly decreased but progesterone significantly increased by administering of cloprostenol concomitant with HCG. It is suggested that cloprostenol was more effective in terminating pregnancy than a combination of cloprostenol and HCG in the rat.
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부화계태아의 수분대사에 관한 연구 - I. 부화일령에 따른 계태야의 수분분할
김영홍,Kim Young-Hong 대한수의사회 1980 대한수의사회지 Vol.16 No.4
The studies were undertaken with the objective to observe water distribution and its content in egg during the incubation periods by dry method. The resalts obtained were sammarized as follows: 1. Chick embryo was not recogenizable on second and third day
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GnRH를 단독으로 또는 PMSG와 동시에 투여했을 때 임신 랫드의 태아에 미치는 영향
김영홍,이근우,손창호 한국임상수의학회 2002 한국임상수의학회지 Vol.19 No.3
The effect of GnRH alone and concomitant with PMSG on the prevention of implantation, termination of pregnancy, and concentration of plasma progesterone were studied in pregnant rats. GnRH 50, 100 or 200mg alone and concomitant with PMSG 25 or 50IU were administered once on day 2 or 9 of gestation, respectively. Rats were autopsied on days 7 or 20. Administration of GnRH on day 2 did not result in the prevention of implantation and termination of pregnancy but resulted in termination of pregnancy administering on day 9. Administration of GnRH concomitant with PMSG on day 2 or 9 resulted in prevention of implantation and termination of pregnancy, but injection of GnRH 50mg concomitant with PMSG 25 IU on day 9 had only one live fetus. Administration of GnRH alone and concomitant with PMSG on day 2 had no effect on the concentration of plasma progesterone determining on day 7. Administration of GnRH concomitant with PMSG on day 2 resulted in decrease of progesterone level determining on day 20 but GnRH alone was normal level. Administration of GnRH alone and concomitant with PMSG on day 9 resulted in decrease of the concentration of progesterone but was normal concentration administering GnRH 50mg concomitant with PMSG 25IU.
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