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- 저자 : Rahbar Amir Hossein (검색결과 2 건)
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Amir Eshraghi,Ali Akbar Mirzaei,Rahbar Rahimi,Hossein Atashi 한국화학공학회 2020 Korean Journal of Chemical Engineering Vol.37 No.10
Mass transfer limitations and kinetics studies were performed for Fischer-Tropsch Synthesis over spherical 10 wt% Fe-10wt% Co-0.5 wt% Pt/79.5 wt% -Al2O3 catalyst in a fixed bed reactor. The external mass transfer limitation was checked by studying the effect of gas hourly space velocity (GHSV) and feed flow rate (at constant GHSV) on CO conversion. Theoretical and practical methods were applied to assess the effect of catalyst pellet size on the internal mass transfer limitation. The results indicated there is external diffusion limitation for GHSV lower than 4,200 h1. Both the theoretical and practical methods showed that the reaction is free of internal diffusion limitation with average particle sizes of 0.21 and 0.42 mm due to Thiele modulus smaller than 0.4, denoting that the rate of reaction is kinetically controlled. The kinetics results demonstrated the combined enol and carbide mechanism-based model was able to provide a good fit for the experimental data.
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The Effects of Resveratrol on Silica-Induced Lung Oxidative Stress and Inflammation in Rat
Esfahani Maryam,Rahbar Amir Hossein,Asl Sara Soleimani,Bashirian Saed,Mir Moeini Effat Sadat,Mehri Fereshteh 한국산업안전보건공단 산업안전보건연구원 2023 Safety and health at work Vol.14 No.1
Background: Chronic exposure to silica is related with the provocation of an inflammatory response and oxidative stress mechanism. Vitamin D has multiple benefits in biological activities particularly respiratory system disease. Method: In this research, 20 male Wistar rats were randomly allocated into four groups (5 rats /group) as follow:Group1 received saline as (negative control) group. The group 2 received a single IT instillation of silica (positive control) group; the group 3was co-administratedwith single ITsilica and Vitamin D (20mg/kg/day) daily for a period of 90 days. The rats of group 4 received Vitamin D daily for a period of 90 days. Results: Silica significantly increased serum and lung total Oxidant Status (TOS). Meanwhile, silica reduced serum and lung total antioxidant capacity (TAC), GSH and tumor necrosis factor-a (TNF-a). Vitamin D treatment meaningfully reversed oxidative stress, antioxidants status and inflammatory response. Also, Vitamin D improved histopathological changes caused by silica. Conclusion: These findings indicate that Vitamin D exerts protective effects against silica-induced lung injury. It seems that Vitamin D has potential use as a therapeutic object for silica induced lung injure.
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